What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Cerebral cortex, Pathology, Neuroscience, Cholinergic and Basal forebrain.
His research in Cerebral cortex intersects with topics in Extracellular matrix component, Biotinylation, Perineuronal net and Cortex.
His work on Alzheimer's disease and Substantia nigra as part of his general Pathology study is frequently connected to Apolipoprotein B, thereby bridging the divide between different branches of science.
His Neuroscience study combines topics from a wide range of disciplines, such as Extracellular and Matrix.
Internal medicine and Endocrinology are the main areas of his Cholinergic studies.
His research integrates issues of Nerve growth factor, AMPA receptor, Parkinson's disease and Cholinergic neuron in his study of Basal forebrain.
His most cited work include:
- Loss of neurons in the nucleus basalis of Meynert in Alzheimer's disease, paralysis agitans and Korsakoff's Disease. (714 citations)
- Neuronal loss in different parts of the nucleus basalis is related to neuritic plaque formation in cortical target areas in alzheimer's disease (372 citations)
- Plastic Neuronal Remodeling Is Impaired in Patients with Alzheimer’s Disease Carrying Apolipoprotein ε4 Allele (290 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Neuroscience, Cholinergic, Basal forebrain, Endocrinology and Internal medicine.
As a part of the same scientific family, Volker Bigl mostly works in the field of Neuroscience, focusing on GABAergic and, on occasion, GABA receptor.
His biological study spans a wide range of topics, including Cerebral cortex, Lesion and Hippocampus.
His Cerebral cortex study integrates concerns from other disciplines, such as Cortex and Pathology.
His Basal forebrain research incorporates elements of Nerve growth factor, Cell biology and Cholinergic neuron.
In his study, Glycolysis is inextricably linked to Temporal cortex, which falls within the broad field of Internal medicine.
He most often published in these fields:
- Neuroscience (40.37%)
- Cholinergic (34.86%)
- Basal forebrain (34.86%)
What were the highlights of his more recent work (between 2000-2005)?
- Amyloid precursor protein (19.27%)
- Cell biology (18.35%)
- Endocrinology (34.86%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Amyloid precursor protein, Cell biology, Endocrinology, Internal medicine and Molecular biology.
Volker Bigl has researched Amyloid precursor protein in several fields, including Genetically modified mouse, Protein kinase C and Gene, Gene isoform.
Volker Bigl interconnects Protein expression, Neuroscience research, Neuroscience, Neurite and Guinea pig in the investigation of issues within Cell biology.
Volker Bigl combines subjects such as Phosphofructokinase and Nerve growth factor with his study of Endocrinology.
His Hippocampus study incorporates themes from Cerebral cortex, Lesion, Cholinergic neuron and Basal forebrain.
His Alzheimer's disease study is associated with Pathology.
Between 2000 and 2005, his most popular works were:
- Astrocytic expression of the Alzheimer's disease β-secretase (BACE1) is stimulus-dependent (133 citations)
- Neuronal and glial β‐secretase (BACE) protein expression in transgenic Tg2576 mice with amyloid plaque pathology (102 citations)
- Subcellular localization suggests novel functions for prolyl endopeptidase in protein secretion. (87 citations)
In his most recent research, the most cited papers focused on:
Volker Bigl mainly focuses on Amyloid precursor protein, Subcellular localization, Prolyl endopeptidase, Alzheimer's disease and Molecular biology.
The concepts of his Subcellular localization study are interwoven with issues in Tubulin, Intracellular and Secretion.
His Prolyl endopeptidase research includes themes of Cerebellum, Extracellular, Hippocampal formation and Transgene.
Endocrinology and Internal medicine are the main topics of his Cerebellum study.
His research on Alzheimer's disease concerns the broader Pathology.
His work carried out in the field of Molecular biology brings together such families of science as Gene, Coding region and DNA.
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