His primary areas of investigation include Cerebral cortex, Pathology, Neuroscience, Cholinergic and Basal forebrain. His research in Cerebral cortex intersects with topics in Extracellular matrix component, Biotinylation, Perineuronal net and Cortex. His work on Alzheimer's disease and Substantia nigra as part of his general Pathology study is frequently connected to Apolipoprotein B, thereby bridging the divide between different branches of science.
His Neuroscience study combines topics from a wide range of disciplines, such as Extracellular and Matrix. Internal medicine and Endocrinology are the main areas of his Cholinergic studies. His research integrates issues of Nerve growth factor, AMPA receptor, Parkinson's disease and Cholinergic neuron in his study of Basal forebrain.
His scientific interests lie mostly in Neuroscience, Cholinergic, Basal forebrain, Endocrinology and Internal medicine. As a part of the same scientific family, Volker Bigl mostly works in the field of Neuroscience, focusing on GABAergic and, on occasion, GABA receptor. His biological study spans a wide range of topics, including Cerebral cortex, Lesion and Hippocampus.
His Cerebral cortex study integrates concerns from other disciplines, such as Cortex and Pathology. His Basal forebrain research incorporates elements of Nerve growth factor, Cell biology and Cholinergic neuron. In his study, Glycolysis is inextricably linked to Temporal cortex, which falls within the broad field of Internal medicine.
His primary scientific interests are in Amyloid precursor protein, Cell biology, Endocrinology, Internal medicine and Molecular biology. Volker Bigl has researched Amyloid precursor protein in several fields, including Genetically modified mouse, Protein kinase C and Gene, Gene isoform. Volker Bigl interconnects Protein expression, Neuroscience research, Neuroscience, Neurite and Guinea pig in the investigation of issues within Cell biology.
Volker Bigl combines subjects such as Phosphofructokinase and Nerve growth factor with his study of Endocrinology. His Hippocampus study incorporates themes from Cerebral cortex, Lesion, Cholinergic neuron and Basal forebrain. His Alzheimer's disease study is associated with Pathology.
Volker Bigl mainly focuses on Amyloid precursor protein, Subcellular localization, Prolyl endopeptidase, Alzheimer's disease and Molecular biology. The concepts of his Subcellular localization study are interwoven with issues in Tubulin, Intracellular and Secretion. His Prolyl endopeptidase research includes themes of Cerebellum, Extracellular, Hippocampal formation and Transgene.
Endocrinology and Internal medicine are the main topics of his Cerebellum study. His research on Alzheimer's disease concerns the broader Pathology. His work carried out in the field of Molecular biology brings together such families of science as Gene, Coding region and DNA.
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Loss of neurons in the nucleus basalis of Meynert in Alzheimer's disease, paralysis agitans and Korsakoff's Disease.
T Arendt;V Bigl;A Arendt;A Tennstedt.
Acta Neuropathologica (1983)
Neuronal loss in different parts of the nucleus basalis is related to neuritic plaque formation in cortical target areas in alzheimer's disease
T. Arendt;V. Bigl;A. Tennstedt;A. Arendt.
Plastic Neuronal Remodeling Is Impaired in Patients with Alzheimer’s Disease Carrying Apolipoprotein ε4 Allele
Thomas Arendt;Cornelia Schindler;Martina K. Brückner;Klaus Eschrich.
The Journal of Neuroscience (1997)
Changes in acetylcholinesterase and butyrylcholinesterase in Alzheimer's disease resemble embryonic development--a study of molecular forms.
Thomas Arendt;Martina K. Brückner;Matthias Lange;Volker Bigl.
Neurochemistry International (1992)
The regulation of amyloid precursor protein metabolism by cholinergic mechanisms and neurotrophin receptor signaling.
Steffen Roßner;Uwe Ueberham;Reinhard Schliebs;J. Regino Perez-Polo.
Progress in Neurobiology (1998)
Systemic administration of defined extracts from Withania somnifera (Indian Ginseng) and shilajit differentially affects cholinergic but not glutamatergic and gabaergic markers in rat brain
Reinhard Schliebs;André Liebmann;Salil K. Bhattacharya;Ashok Kumar.
Neurochemistry International (1997)
Astrocytic expression of the Alzheimer's disease β-secretase (BACE1) is stimulus-dependent
Maike Hartlage-Rübsamen;Ulrike Zeitschel;Jenny Apelt;Ulrich Gärtner.
Activities of key glycolytic enzymes in the brains of patients with Alzheimer's disease.
M. Bigl;M. K. Brückner;T. Arendt;V. Bigl.
Journal of Neural Transmission (1999)
Chondroitin sulfate proteoglycan-immunoreactivity of lectin-labeled perineuronal nets around parvalbumin-containing neurons.
Wolfgang Ha¨rtig;Kurt Brauer;Volker Bigl;Gert Bru¨ckner.
Brain Research (1994)
Cortical areas are revealed by distribution patterns of proteoglycan components and parvalbumin in the Mongolian gerbil and rat.
Gert Brückner;Gudrun Seeger;Kurt Brauer;Wolfgang Härtig.
Brain Research (1994)
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