What is he best known for?
The fields of study he is best known for:
- Gene
- Central nervous system
- Internal medicine
His primary areas of study are Retina, Cell biology, Microglia, Giant retinal ganglion cells and Neuroglia.
His Retina study combines topics in areas such as Anatomy and Optic nerve.
The various areas that V.H. Perry examines in his Microglia study include Choroid plexus, Pathology, Macrophage and Complement receptor.
V.H. Perry has included themes like Intrinsically photosensitive retinal ganglion cells and Ganglion in his Giant retinal ganglion cells study.
His study with Neuroglia involves better knowledge in Central nervous system.
He interconnects Blood–brain barrier and In vivo in the investigation of issues within Immunology.
His most cited work include:
- Heterogeneity in the distribution and morphology of microglia in the normal adult mouse brain (1396 citations)
- Immunohistochemical localization of macrophages and microglia in the adult and developing mouse brain. (782 citations)
- Retinal ganglion cells that project to the dorsal lateral geniculate nucleus in the macaque monkey (603 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Immunology, Retina, Pathology, Microglia and Ganglion.
His Immunology research is multidisciplinary, relying on both Macrophage, Central nervous system, Blood–brain barrier and Parenchyma.
In his study, Lateral geniculate nucleus is inextricably linked to Anatomy, which falls within the broad field of Retina.
His Pathology research incorporates themes from Lipopolysaccharide and Antigen.
His Microglia study also includes
- Neuroglia that intertwine with fields like Neuroinflammation,
- Cell biology that intertwine with fields like Antibody.
His study in Ganglion is interdisciplinary in nature, drawing from both Thalamus, Retinal ganglion cell and Retinal ganglion.
He most often published in these fields:
- Immunology (36.36%)
- Retina (32.47%)
- Pathology (32.47%)
What were the highlights of his more recent work (between 1996-1999)?
- Immunology (36.36%)
- Microglia (31.17%)
- Parenchyma (14.29%)
In recent papers he was focusing on the following fields of study:
V.H. Perry mostly deals with Immunology, Microglia, Parenchyma, Pathology and Scrapie.
Immunology is often connected to Blood–brain barrier in his work.
The Microglia study combines topics in areas such as Lesion, Retinal ganglion cell, Molecular biology, Antibody and Macrophage.
His biological study spans a wide range of topics, including Inflammation, Myelin, Central nervous system and Immunohistochemistry.
His work on Infiltration as part of general Pathology study is frequently linked to Mouse hepatitis virus, bridging the gap between disciplines.
The concepts of his In vivo study are interwoven with issues in Endothelial stem cell and Cell biology.
Between 1996 and 1999, his most popular works were:
- Loss of the tight junction proteins occludin and zonula occludens-1 from cerebral vascular endothelium during neutrophil-induced blood–brain barrier breakdown in vivo (317 citations)
- Matrix metalloproteinase expression in an experimentally-induced DTH model of multiple sclerosis in the rat CNS. (162 citations)
- CXC chemokines generate age-related increases in neutrophil-mediated brain inflammation and blood–brain barrier breakdown (136 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Genetics
V.H. Perry mainly investigates Parenchyma, Immunology, Blood–brain barrier, Rodent and Central nervous system.
His Parenchyma research is multidisciplinary, incorporating perspectives in Downregulation and upregulation, Molecular biology, Myelin, Messenger RNA and Matrix metalloproteinase.
His Immunology study combines topics in areas such as Endothelium, Tight junction, Cell biology and In vivo.
His Blood–brain barrier research is multidisciplinary, relying on both Endothelial stem cell, Occludin and Cell junction.
His Rodent study spans across into subjects like Chemotaxis, Brain damage, Inflammation and Cytokine.
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