His primary areas of study are Macular degeneration, Pathology, Genetics, Retina and Retinal pigment epithelium. His research on Macular degeneration focuses in particular on Drusen. The study incorporates disciplines such as Basal lamina and Inflammation in addition to Drusen.
In Pathology, Robert F. Mullins works on issues like Complement system, which are connected to Pathogenesis and Glomerulonephritis. He is studying Retinal degeneration, which is a component of Retina. The Anatomy study combines topics in areas such as Ophthalmology and Choroidal neovascularization.
Robert F. Mullins focuses on Macular degeneration, Retina, Retinal, Pathology and Ophthalmology. He mostly deals with Drusen in his studies of Macular degeneration. His Retina study combines topics in areas such as Anatomy and Cell biology.
His Cell biology study integrates concerns from other disciplines, such as Endothelial stem cell and Gene. His Retinal research is multidisciplinary, incorporating perspectives in Cell and Gene expression. He specializes in Ophthalmology, namely Optical coherence tomography.
Cell biology, Retinal, Retina, Gene and Macular degeneration are his primary areas of study. His study in Cell biology is interdisciplinary in nature, drawing from both Endothelial stem cell, Rat retina, Cell type and Immune system. His Retinal research is multidisciplinary, incorporating elements of Oxidative stress, CRISPR and Perfusion.
His Retina research incorporates elements of RNA, Transcriptome, Gene expression and Pathology. His work carried out in the field of Pathology brings together such families of science as Geographic atrophy, Choroid and Complement system. Ophthalmology covers Robert F. Mullins research in Macular degeneration.
His scientific interests lie mostly in Cell biology, Retina, Retinal, Gene and Retinal degeneration. The Retina study which covers Transcriptome that intersects with RNA and Pathology. His Pathology research is multidisciplinary, relying on both Retinal pigment epithelium, Choroid and Geographic atrophy.
As part of one scientific family, Robert F. Mullins deals mainly with the area of Retinal, narrowing it down to issues related to the Cell, and often Frameshift mutation, Transcription factor, CRISPR and Photoreceptor cell. His Gene research integrates issues from Rat retina and Inflammation. Robert F. Mullins is involved in the study of Macular degeneration that focuses on Drusen in particular.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
An Integrated Hypothesis That Considers Drusen as Biomarkers of Immune-Mediated Processes at the RPE-Bruch's Membrane Interface in Aging and Age-Related Macular Degeneration
Gregory S. Hageman;Phil J. Luthert;N.H. Victor Chong;N.H. Victor Chong;Lincoln V. Johnson.
Progress in Retinal and Eye Research (2001)
A role for local inflammation in the formation of drusen in the aging eye.
Don H Anderson;Robert F Mullins;Gregory S Hageman;Lincoln V Johnson.
American Journal of Ophthalmology (2002)
Drusen associated with aging and age-related macular degeneration contain proteins common to extracellular deposits associated with atherosclerosis, elastosis, amyloidosis, and dense deposit disease
Robert F. Mullins;Stephen R. Russell;Don H. Anderson;Gregory S. Hageman.
The FASEB Journal (2000)
T-cell immunoglobulin and mucin domain 1 (TIM-1) is a receptor for Zaire Ebolavirus and Lake Victoria Marburgvirus
Andrew S Kondratowicz;Nicholas J Lennemann;Patrick L Sinn;Robert A Davey.
Proceedings of the National Academy of Sciences of the United States of America (2011)
Bbs2-null mice have neurosensory deficits, a defect in social dominance, and retinopathy associated with mislocalization of rhodopsin
Darryl Y. Nishimura;Melissa Fath;Robert F. Mullins;Charles Searby.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Retinal neurodegeneration may precede microvascular changes characteristic of diabetic retinopathy in diabetes mellitus.
Elliott H. Sohn;Hille W. van Dijk;Chunhua Jiao;Pauline H. B. Kok.
Proceedings of the National Academy of Sciences of the United States of America (2016)
Bardet-Biedl syndrome type 4 (BBS4)-null mice implicate Bbs4 in flagella formation but not global cilia assembly.
Kirk Mykytyn;Robert F. Mullins;Michael Andrews;Annie P. Chiang.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Choriocapillaris vascular dropout related to density of drusen in human eyes with early age-related macular degeneration.
Robert F. Mullins;Micaela N. Johnson;Elizabeth A. Faidley;Jessica M. Skeie.
Investigative Ophthalmology & Visual Science (2011)
Non-secretion of mutant proteins of the glaucoma gene myocilin in cultured trabecular meshwork cells and in aqueous humor
Nasreen Jacobson;Michael Andrews;Allan R. Shepard;Darryl Nishimura.
Human Molecular Genetics (2001)
Mutations in the novel protocadherin PCDH15 cause Usher syndrome type 1F
Kumar N. Alagramam;Huijun Yuan;Markus H. Kuehn;Crystal L. Murcia.
Human Molecular Genetics (2001)
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