D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 72 Citations 17,552 390 World Ranking 16436 National Ranking 8374
Biology and Biochemistry D-index 72 Citations 17,419 368 World Ranking 3963 National Ranking 2006

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Mutation

His primary areas of study are Macular degeneration, Pathology, Genetics, Retina and Retinal pigment epithelium. His research on Macular degeneration focuses in particular on Drusen. The study incorporates disciplines such as Basal lamina and Inflammation in addition to Drusen.

In Pathology, Robert F. Mullins works on issues like Complement system, which are connected to Pathogenesis and Glomerulonephritis. He is studying Retinal degeneration, which is a component of Retina. The Anatomy study combines topics in areas such as Ophthalmology and Choroidal neovascularization.

His most cited work include:

  • An Integrated Hypothesis That Considers Drusen as Biomarkers of Immune-Mediated Processes at the RPE-Bruch's Membrane Interface in Aging and Age-Related Macular Degeneration (1089 citations)
  • A role for local inflammation in the formation of drusen in the aging eye. (905 citations)
  • Drusen associated with aging and age-related macular degeneration contain proteins common to extracellular deposits associated with atherosclerosis, elastosis, amyloidosis, and dense deposit disease (763 citations)

What are the main themes of his work throughout his whole career to date?

Robert F. Mullins focuses on Macular degeneration, Retina, Retinal, Pathology and Ophthalmology. He mostly deals with Drusen in his studies of Macular degeneration. His Retina study combines topics in areas such as Anatomy and Cell biology.

His Cell biology study integrates concerns from other disciplines, such as Endothelial stem cell and Gene. His Retinal research is multidisciplinary, incorporating perspectives in Cell and Gene expression. He specializes in Ophthalmology, namely Optical coherence tomography.

He most often published in these fields:

  • Macular degeneration (24.43%)
  • Retina (24.94%)
  • Retinal (23.68%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (18.14%)
  • Retinal (23.68%)
  • Retina (24.94%)

In recent papers he was focusing on the following fields of study:

Cell biology, Retinal, Retina, Gene and Macular degeneration are his primary areas of study. His study in Cell biology is interdisciplinary in nature, drawing from both Endothelial stem cell, Rat retina, Cell type and Immune system. His Retinal research is multidisciplinary, incorporating elements of Oxidative stress, CRISPR and Perfusion.

His Retina research incorporates elements of RNA, Transcriptome, Gene expression and Pathology. His work carried out in the field of Pathology brings together such families of science as Geographic atrophy, Choroid and Complement system. Ophthalmology covers Robert F. Mullins research in Macular degeneration.

Between 2018 and 2021, his most popular works were:

  • Single-cell transcriptomics of the human retinal pigment epithelium and choroid in health and macular degeneration. (45 citations)
  • Molecular characterization of foveal versus peripheral human retina by single-cell RNA sequencing. (39 citations)
  • Choriocapillaris Degeneration in Geographic Atrophy. (20 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Mutation

His scientific interests lie mostly in Cell biology, Retina, Retinal, Gene and Retinal degeneration. The Retina study which covers Transcriptome that intersects with RNA and Pathology. His Pathology research is multidisciplinary, relying on both Retinal pigment epithelium, Choroid and Geographic atrophy.

As part of one scientific family, Robert F. Mullins deals mainly with the area of Retinal, narrowing it down to issues related to the Cell, and often Frameshift mutation, Transcription factor, CRISPR and Photoreceptor cell. His Gene research integrates issues from Rat retina and Inflammation. Robert F. Mullins is involved in the study of Macular degeneration that focuses on Drusen in particular.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

An Integrated Hypothesis That Considers Drusen as Biomarkers of Immune-Mediated Processes at the RPE-Bruch's Membrane Interface in Aging and Age-Related Macular Degeneration

Gregory S. Hageman;Phil J. Luthert;N.H. Victor Chong;N.H. Victor Chong;Lincoln V. Johnson.
Progress in Retinal and Eye Research (2001)

1499 Citations

A role for local inflammation in the formation of drusen in the aging eye.

Don H Anderson;Robert F Mullins;Gregory S Hageman;Lincoln V Johnson.
American Journal of Ophthalmology (2002)

1288 Citations

Drusen associated with aging and age-related macular degeneration contain proteins common to extracellular deposits associated with atherosclerosis, elastosis, amyloidosis, and dense deposit disease

Robert F. Mullins;Stephen R. Russell;Don H. Anderson;Gregory S. Hageman.
The FASEB Journal (2000)

820 Citations

T-cell immunoglobulin and mucin domain 1 (TIM-1) is a receptor for Zaire Ebolavirus and Lake Victoria Marburgvirus

Andrew S Kondratowicz;Nicholas J Lennemann;Patrick L Sinn;Robert A Davey.
Proceedings of the National Academy of Sciences of the United States of America (2011)

402 Citations

Bbs2-null mice have neurosensory deficits, a defect in social dominance, and retinopathy associated with mislocalization of rhodopsin

Darryl Y. Nishimura;Melissa Fath;Robert F. Mullins;Charles Searby.
Proceedings of the National Academy of Sciences of the United States of America (2004)

390 Citations

Retinal neurodegeneration may precede microvascular changes characteristic of diabetic retinopathy in diabetes mellitus.

Elliott H. Sohn;Hille W. van Dijk;Chunhua Jiao;Pauline H. B. Kok.
Proceedings of the National Academy of Sciences of the United States of America (2016)

385 Citations

Bardet-Biedl syndrome type 4 (BBS4)-null mice implicate Bbs4 in flagella formation but not global cilia assembly.

Kirk Mykytyn;Robert F. Mullins;Michael Andrews;Annie P. Chiang.
Proceedings of the National Academy of Sciences of the United States of America (2004)

346 Citations

Choriocapillaris vascular dropout related to density of drusen in human eyes with early age-related macular degeneration.

Robert F. Mullins;Micaela N. Johnson;Elizabeth A. Faidley;Jessica M. Skeie.
Investigative Ophthalmology & Visual Science (2011)

338 Citations

Non-secretion of mutant proteins of the glaucoma gene myocilin in cultured trabecular meshwork cells and in aqueous humor

Nasreen Jacobson;Michael Andrews;Allan R. Shepard;Darryl Nishimura.
Human Molecular Genetics (2001)

329 Citations

Mutations in the novel protocadherin PCDH15 cause Usher syndrome type 1F

Kumar N. Alagramam;Huijun Yuan;Markus H. Kuehn;Crystal L. Murcia.
Human Molecular Genetics (2001)

323 Citations

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