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Biology and Biochemistry

D-Index
67
Citations
16250
World Ranking
8215
National Ranking
601

Overview

Michael Naumann is affiliated with Otto-von-Guericke University Magdeburg in Germany. Their research primarily spans the fields of Biochemistry, Genetics and Molecular Biology, with a significant contribution to Medicine. The subfields of study in which they have been active include Molecular Biology, Oncology, Surgery, Cancer Research, and Immunology.

Their work covers several key topics related to cellular and molecular processes, including:

  • Ubiquitin and proteasome pathways
  • Helicobacter pylori-related gastroenterology studies
  • NF-κB signaling pathways
  • Galectins and cancer biology
  • Peptidase inhibition and analysis
  • Cancer-related molecular pathways
  • Protein degradation and inhibitors

Frequent co-authors collaborating with Michael Naumann include:

  • Gunter Maubach
  • Eric Schulze
  • Matthias Stein
  • Supattra Chaithongyot
  • Olga Sokolova

The scientist publishes regularly in specific academic venues, most notably:

  • Trends in Molecular Medicine
  • Cellular and Molecular Life Sciences
  • Biomolecules
  • EMBO Reports
  • Cell Death and Differentiation

Recent papers authored or co-authored by Michael Naumann include:

  • The COP9 Signalosome: A Multi-DUB Complex, 2020, Biomolecules
  • Peptide microarray-based analysis of antibody responses to SARS-CoV-2 identifies unique epitopes with potential for diagnostic test development, 2021, European Journal of Immunology
  • The deubiquitinase OTUB1 augments NF-κB-dependent immune responses in dendritic cells in infection and inflammation by stabilizing UBC13, 2020, Cellular and Molecular Immunology
  • Helicobacter pylori-induced NF-κB: trailblazer for gastric pathophysiology, 2022, Trends in Molecular Medicine
  • Matrix Metalloproteinases in Helicobacter pylori-Associated Gastritis and Gastric Cancer, 2022, International Journal of Molecular Sciences

Best Publications

  • Translocation of the Helicobacter pylori CagA protein in gastric epithelial cells by a type IV secretion apparatus.

    Steffen Backert;Elke Ziska;Volker Brinkmann;Ursula Zimny-Arndt

  • A novel protein complex involved in signal transduction possessing similarities to 26S proteasome subunits

    Michael Seeger;Regine Kraft;Katherine Ferrell;Dawadschargal Bech-Otschir

  • Activation of NF-kappa B in vivo is regulated by multiple phosphorylations.

    M Naumann;C Scheidereit

  • Helicobacter pylori CagA protein targets the c-Met receptor and enhances the motogenic response

    Yuri Churin;Laila Al-Ghoul;Oliver Kepp;Thomas F. Meyer

  • The ankyrin repeat domains of the NF-kappa B precursor p105 and the protooncogene bcl-3 act as specific inhibitors of NF-kappa B DNA binding

    E N Hatada;A Nieters;F G Wulczyn;M Naumann

  • Beyond IkappaBs: alternative regulation of NF-kappaB activity.

    Manfred Neumann;Michael Naumann

  • Helicobacter pylori eradication has the potential to prevent gastric cancer : a state-of-the-art critique

    Peter Malfertheiner;Pentti Sipponen;Michael Naumann;Paul Moayyedi

  • Candidate proto-oncogene bcl-3 encodes a subunit-specific inhibitor of transcription factor NF-κB

    Wulczyn Fg;Naumann M;Scheidereit C

  • Inhibition of p38 MAP kinase-and RICK/NF-κB-signaling suppresses inflammatory bowel disease

    Eike Hollenbach;Manfred Neumann;Michael Vieth;Albert Roessner

  • Neisserial porin (PorB) causes rapid calcium influx in target cells and induces apoptosis by the activation of cysteine proteases

    Anne Müller;Dirk Günther;Frank Düx;Michael Naumann

  • CSN controls NF-κB by deubiquitinylation of IκBα

    Katrin Schweitzer;Przemyslaw M Bozko;Wolfgang Dubiel;Michael Naumann

  • Pathogenesis of Helicobacter pylori infection

    Steffen Backert;Matthias Neddermann;Gunter Maubach;Michael Naumann

  • Activation of activator protein 1 and stress response kinases in epithelial cells colonized by Helicobacter pylori encoding the cag pathogenicity island.

    Michael Naumann;Silja Wessler;Cornelia Bartsch;Björn Wieland

  • Unified nomenclature for the COP9 signalosome and its subunits: an essential regulator of development

    XW Deng;W Dubiel;N Wei;K Hofmann

  • Immunohistochemical and electron microscopic study of interaction of Yersinia enterocolitica serotype O8 with intestinal mucosa during experimental enteritis.

    C Hanski;U Kutschka;H P Schmoranzer;M Naumann

  • What a disorder: proinflammatory signaling pathways induced by Helicobacter pylori.

    Steffen Backert;Michael Naumann

  • Fission Yeast COP9/Signalosome Suppresses Cullin Activity through Recruitment of the Deubiquitylating Enzyme Ubp12p

    Chunshui Zhou;Susan Wee;Edward Rhee;Michael Naumann

  • Inhibition of RICK/Nuclear Factor-κB and p38 Signaling Attenuates the Inflammatory Response in a Murine Model of Crohn Disease

    Eike Hollenbach;Michael Vieth;Albert Roessner;Manfred Neumann

  • Encoding and transducing the synaptic or extrasynaptic origin of NMDA receptor signals to the nucleus.

    Anna Karpova;Marina Mikhaylova;Marina Mikhaylova;Sujoy Bera;Julia Bär

  • The NF-kappa B precursor p105 and the proto-oncogene product Bcl-3 are I kappa B molecules and control nuclear translocation of NF-kappa B.

    M Naumann;F G Wulczyn;C Scheidereit

Frequent Co-Authors

Thomas F. Meyer
Thomas F. Meyer Max Planck Institute for Infection Biology
Dirk Schlüter
Dirk Schlüter Otto-von-Guericke University Magdeburg
Eckart D. Gundelfinger
Eckart D. Gundelfinger Leibniz Institute for Neurobiology
Peter Hauptmann
Peter Hauptmann Otto-von-Guericke University Magdeburg
Steffen Backert
Steffen Backert University of Erlangen-Nuremberg
Bertram Wiedenmann
Bertram Wiedenmann Charité - University Medicine Berlin
Peter Malfertheiner
Peter Malfertheiner Otto-von-Guericke University Magdeburg
Claus Scheidereit
Claus Scheidereit Max Delbrück Center for Molecular Medicine
Uwe Lendeckel
Uwe Lendeckel Greifswald University Hospital
Michael Vieth
Michael Vieth University of Erlangen-Nuremberg

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