What is he best known for?
The fields of study he is best known for:
Cell biology, Transcription factor, Molecular biology, Helicobacter pylori and Signal transduction are his primary areas of study.
Michael Naumann studies Phosphorylation, a branch of Cell biology.
His Transcription factor study focuses on DNA-binding protein in particular.
His Molecular biology study combines topics from a wide range of disciplines, such as Proteases, Apoptosis and Protein subunit.
He has researched Helicobacter pylori in several fields, including CagA, Cancer, Pathogenicity island and Microbiology.
His Signal transduction research incorporates elements of Proinflammatory cytokine, Inflammation and Immunology.
His most cited work include:
- Translocation of the Helicobacter pylori CagA protein in gastric epithelial cells by a type IV secretion apparatus. (359 citations)
- Activation of NF-kappa B in vivo is regulated by multiple phosphorylations. (338 citations)
- A novel protein complex involved in signal transduction possessing similarities to 26S proteasome subunits (327 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Cell biology, Molecular biology, Cancer research, Helicobacter pylori and Immunology.
His work deals with themes such as Ubiquitin and Transcription factor, which intersect with Cell biology.
His work carried out in the field of Molecular biology brings together such families of science as Gene expression, NFKB1, Apoptosis, Protein subunit and Cytotoxic T cell.
In his research, Regulation of gene expression is intimately related to MAPK/ERK pathway, which falls under the overarching field of Cancer research.
The concepts of his Helicobacter pylori study are interwoven with issues in Secretion, Microbiology and CagA.
His research on Proinflammatory cytokine also deals with topics like
- Kinase which is related to area like Phosphorylation,
- Cytokine together with Chemokine.
He most often published in these fields:
- Cell biology (33.15%)
- Molecular biology (23.37%)
- Cancer research (20.65%)
What were the highlights of his more recent work (between 2017-2021)?
- Cell biology (33.15%)
- Cancer research (20.65%)
- Ubiquitin (9.24%)
In recent papers he was focusing on the following fields of study:
Michael Naumann mainly focuses on Cell biology, Cancer research, Ubiquitin, Helicobacter pylori and Kinase.
His Cell biology study incorporates themes from Tumor necrosis factor alpha, Neuroplastin and Interactome.
His Cancer research research integrates issues from Carcinogenesis, Cancer, Cancer cell and Inflammation.
Michael Naumann interconnects Enzyme, Active site and NF-κB, Signal transduction in the investigation of issues within Ubiquitin.
In his research on the topic of Helicobacter pylori, CagA is strongly related with Secretion.
Michael Naumann combines subjects such as Bacterial adhesin, RNA interference, Molecular biology and Microbiology with his study of CagA.
Between 2017 and 2021, his most popular works were:
- Cullin 3-Based Ubiquitin Ligases as Master Regulators of Mammalian Cell Differentiation. (34 citations)
- Influence of eukaryotic translation initiation factor 6 on non–small cell lung cancer development and progression (14 citations)
- HopQ impacts the integrin α5β1-independent NF-κB activation by Helicobacter pylori in CEACAM expressing cells (9 citations)
In his most recent research, the most cited papers focused on:
Michael Naumann mainly investigates Cancer research, Carcinogenesis, Cell biology, Helicobacter pylori and Cancer.
His Cancer research study combines topics in areas such as Caspase 3, Gastritis, Camptothecin, Gastric mucosa and DNA repair.
His study in Carcinogenesis is interdisciplinary in nature, drawing from both Viability assay, MTT assay, Cancer cell and Caspase 8.
Specifically, his work in Cell biology is concerned with the study of NF-κB.
His Helicobacter pylori research incorporates themes from Inflammation, Secretion and Metaplasia.
His biological study spans a wide range of topics, including Transcript level and Kinase, NF-kappaB-inducing kinase.
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