Genetics, Gene, Locus, Molecular biology and Allele are her primary areas of study. Her Neurofibromatosis, Genetic linkage, Genetic marker, Single-nucleotide polymorphism and Chromosome 17 investigations are all subjects of Genetics research. Her Candidate gene, Somatic cell and Molecular medicine study in the realm of Gene interacts with subjects such as Leukocyte degranulation and SNARE complex.
Her Locus study integrates concerns from other disciplines, such as Susceptibility locus, Generalized vitiligo, Gene mapping and Exon. Her Molecular biology study combines topics in areas such as Positional cloning, GTPase, Complementary DNA, Neurofibromin 1 and Sequence analysis. Her Allele research is multidisciplinary, incorporating perspectives in Mutation, Taste and Receptor.
Her primary areas of study are Genetics, Gene, Neurofibromatosis, Molecular biology and Internal medicine. Genetics is a component of her Chromosome 17, Locus, Genetic marker, Chromosome and Allele studies. Her Gene research includes elements of Elastin and DNA.
Her work carried out in the field of Neurofibromatosis brings together such families of science as Genetic disorder and Germline mutation. Margaret R. Wallace combines subjects such as Complementary DNA and Frameshift mutation with her study of Molecular biology. Her Internal medicine research is multidisciplinary, incorporating elements of Endocrinology and Oncology.
The scientist’s investigation covers issues in Neurofibromatosis, Cancer research, Internal medicine, Neurofibromin 1 and Single-nucleotide polymorphism. Her Neurofibromatosis research also works with subjects such as
Her Single-nucleotide polymorphism study is concerned with Gene in general. Proband is the subject of her research, which falls under Genetics. In the field of Genetics, her study on Mutation overlaps with subjects such as FLNC.
Margaret R. Wallace focuses on Cancer research, Neurofibromatosis, Neurofibroma, Genetics and Malignant transformation. The study incorporates disciplines such as Neurofibromin 1, Malignant peripheral nerve sheath tumor, In vivo and Immortalised cell line in addition to Cancer research. She focuses mostly in the field of Neurofibromatosis, narrowing it down to matters related to Tumor suppressor gene and, in some cases, Exon, Phenocopy, Mutant, Mutation and Café au lait spot.
She has researched Neurofibroma in several fields, including Cell culture, Exome and Cell growth. The various areas that Margaret R. Wallace examines in her Genetics study include Periodontitis, Pathogenesis and Bioinformatics. Her work focuses on many connections between Malignant transformation and other disciplines, such as Germline mutation, that overlap with her field of interest in CDKN2A, Carcinogenesis and Molecular biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A polymorphic DNA marker genetically linked to Huntington's disease
James F. Gusella;Nancy S. Wexler;P. Michael Conneally;Susan L. Naylor.
Nature (1983)
Type 1 neurofibromatosis gene: identification of a large transcript disrupted in three NF1 patients
Margaret R. Wallace;Douglas A. Marchuk;Lone B. Andersen;Roxanne Letcher.
Science (1990)
Identification of the familial cylindromatosis tumour-suppressor gene.
Graham R. Bignell;William Warren;Sheila Seal;Meiko Takahashi.
Nature Genetics (2000)
The melanocortin-1 receptor gene mediates female-specific mechanisms of analgesia in mice and humans
Jeffrey S. Mogil;Sonya G. Wilson;Elissa J. Chesler;Andrew L. Rankin.
Proceedings of the National Academy of Sciences of the United States of America (2003)
A de novo Alu insertion results in neurofibromatosis type 1.
Margaret R. Wallace;Margaret R. Wallace;Lone B. Andersen;Ann M. Saulino;Paula E. Gregory.
Nature (1991)
cDNA cloning of the type 1 neurofibromatosis gene: Complete sequence of the NF1 gene product
Douglas A. Marchuk;Ann M. Saulino;Roxanne Tavakkol;Manju Swaroop.
Genomics (1991)
The A118G single nucleotide polymorphism of the μ-opioid receptor gene (OPRM1) is associated with pressure pain sensitivity in humans
Roger B. Fillingim;Roger B. Fillingim;Lee Kaplan;Roland Staud;Timothy J. Ness.
The Journal of Pain (2005)
Variant of TYR and autoimmunity susceptibility loci in generalized vitiligo.
Ying Jin;Stanca A. Birlea;Pamela R. Fain;Katherine Gowan.
The New England Journal of Medicine (2010)
Genome-wide association analyses identify 13 new susceptibility loci for generalized vitiligo
Ying Jin;Stanca A Birlea;Pamela R Fain;Tracey M Ferrara.
Nature Genetics (2012)
Benign neurofibromas in type 1 neurofibromatosis (NF1) show somatic deletions of the NF1 gene
Steven D. Colman;Charles A. Williams;Margaret R. Wallace.
Nature Genetics (1995)
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