His primary scientific interests are in Biochemistry, Lipid peroxidation, Internal medicine, Endocrinology and Isoprostanes. His research integrates issues of Oxygen tension and In vivo in his study of Biochemistry. His research on Lipid peroxidation concerns the broader Oxidative stress.
His Internal medicine study frequently draws connections between adjacent fields such as Antioxidant. His Endocrinology research integrates issues from Biomarker, Mast cell activation syndrome and Vitamin E. He has included themes like Stereochemistry and Pharmacology in his Isoprostanes study.
His primary areas of investigation include Internal medicine, Biochemistry, Lipid peroxidation, Endocrinology and Oxidative stress. His work deals with themes such as Gastroenterology and Immunology, which intersect with Internal medicine. He focuses mostly in the field of Biochemistry, narrowing it down to topics relating to In vivo and, in certain cases, Eicosanoid.
His study focuses on the intersection of Lipid peroxidation and fields such as Anesthesia with connections in the field of Ischemia. His studies deal with areas such as Inflammation and Alzheimer's disease as well as Endocrinology. His Oxidative stress research incorporates elements of Reactive oxygen species, Mitochondrion, Oxidative phosphorylation and Antioxidant.
L. Jackson Roberts focuses on Internal medicine, Oxidative stress, Endocrinology, Lipid peroxidation and Cell biology. His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology, Surgery and Immunology. The study incorporates disciplines such as Lung injury, Lipid oxidation, Mitochondrion and Arachidonic acid in addition to Oxidative stress.
His study on Lipid peroxidation is covered under Biochemistry. His Biochemistry research is multidisciplinary, relying on both Liquid chromatography–mass spectrometry and Function. The concepts of his Cell biology study are interwoven with issues in Apoptosis, Gene and Disease.
L. Jackson Roberts spends much of his time researching Internal medicine, Oxidative stress, Endocrinology, Biochemistry and Lipid peroxidation. His Internal medicine study incorporates themes from Gastroenterology and Immunology. He combines subjects such as Reactive oxygen species, Hyperoxia, Atrial natriuretic peptide and Downregulation and upregulation with his study of Oxidative stress.
His Endocrinology study combines topics from a wide range of disciplines, such as Arthritis, Inflammation, Erythrocyte sedimentation rate, Tocilizumab and Mitochondrion. His Radical and F2-Isoprostane study in the realm of Biochemistry connects with subjects such as Physiological responses, Terminology and Biochemical engineering. His study in the field of Isoprostanes is also linked to topics like Biological studies.
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Increase in circulating products of lipid peroxidation (F2-isoprostanes) in smokers. Smoking as a cause of oxidative damage.
J D Morrow;B Frei;A W Longmire;J M Gaziano.
The New England Journal of Medicine (1995)
Measuring reactive oxygen and nitrogen species with fluorescent probes: challenges and limitations.
Balaraman Kalyanaraman;Victor Darley-Usmar;Kelvin J.A. Davies;Phyllis A. Dennery.
Free Radical Biology and Medicine (2012)
Isoprostanes: markers and mediators of oxidative stress
Paolo Montuschi;Peter J. Barnes;L. Jackson Roberts.
The FASEB Journal (2004)
The isoprostanes: Unique bioactive products of lipid peroxidation
Jason D. Morrow;L.Jackson Roberts.
Progress in Lipid Research (1997)
CuZnSOD deficiency leads to persistent and widespread oxidative damage and hepatocarcinogenesis later in life
Sailaja Elchuri;Terry D. Oberley;Wenbo Qi;Richard S. Eisenstein.
Noncyclooxygenase oxidative formation of a series of novel prostaglandins: analytical ramifications for measurement of eicosanoids.
Jason D. Morrow;Thomas M. Harris;L. Jackson Roberts.
Analytical Biochemistry (1990)
Lipid peroxidation in aging brain and Alzheimer's disease.
Thomas J Montine;M.Diana Neely;Joseph F Quinn;M.Flint Beal.
Free Radical Biology and Medicine (2002)
Preterm resuscitation with low oxygen causes less oxidative stress, inflammation, and chronic lung disease.
Maximo Vento;Manuel Moro;Raquel Escrig;Luis Arruza.
Absence of CuZn superoxide dismutase leads to elevated oxidative stress and acceleration of age-dependent skeletal muscle atrophy
Florian L. Muller;Wook Song;Yuhong Liu;Asish Chaudhuri;Asish Chaudhuri.
Free Radical Biology and Medicine (2006)
Formation of Isoprostane-like Compounds (Neuroprostanes) in Vivo from Docosahexaenoic Acid
L. Jackson Roberts;Thomas J. Montine;William R. Markesbery;Andrew R. Tapper.
Journal of Biological Chemistry (1998)
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