The scientist’s investigation covers issues in Neuroscience, Postsynaptic potential, Synapse, Long-term depression and Biophysics. His Neuroscience study integrates concerns from other disciplines, such as Long-term potentiation and AMPA receptor. His studies examine the connections between Postsynaptic potential and genetics, as well as such issues in Synaptic plasticity, with regards to Calcium.
In his study, Dendrite, Anatomy and Dendritic filopodia is inextricably linked to Neurotransmission, which falls within the broad field of Synapse. His study looks at the relationship between Biophysics and fields such as NMDA receptor, as well as how they intersect with chemical problems. His work carried out in the field of Depolarization brings together such families of science as Hippocampal formation and Glutamate receptor.
John A. Connor mostly deals with Neuroscience, Biophysics, Cell biology, Hippocampal formation and Glutamate receptor. His research integrates issues of Synaptic plasticity, Long-term potentiation, Long-term depression and Postsynaptic potential in his study of Neuroscience. John A. Connor works mostly in the field of Long-term depression, limiting it down to topics relating to Synapse and, in certain cases, Neurotransmission and AMPA receptor, as a part of the same area of interest.
The Biophysics study combines topics in areas such as Extracellular, Biochemistry, Intracellular and Calcium. He combines subjects such as Excitotoxicity, Electrophysiology and Cerebellum with his study of Cell biology. His study focuses on the intersection of Glutamate receptor and fields such as NMDA receptor with connections in the field of Excitatory postsynaptic potential and In vitro.
John A. Connor focuses on Neuroscience, Glutamate receptor, Entorhinal cortex, Perirhinal cortex and Premovement neuronal activity. John A. Connor has researched Neuroscience in several fields, including Nonsynaptic plasticity and Audiology. His Glutamate receptor research integrates issues from Calcium, Dendrite, Cytosol, Hippocampal formation and Microstimulation.
His study in Calcium is interdisciplinary in nature, drawing from both NMDA receptor, Biophysics, Biochemistry, Intracellular and Hippocampus. Dendrite is closely attributed to Extracellular in his research. His Entorhinal cortex study combines topics in areas such as Dentate gyrus, Subiculum, Reversal potential, Status epilepticus and Inhibitory postsynaptic potential.
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Dendritic spines as individual neuronal compartments for synaptic Ca2+ responses.
Wolfgang Müller;John A. Connor.
Nature (1991)
Targeted disruption of NMDA receptor 1 gene abolishes NMDA response and results in neonatal death
Douglas Forrest;Michisuke Yuzaki;Holly D. Soares;Lily Ng.
Neuron (1994)
A long-term depression of AMPA currents in cultured cerebellar Purkinje neurons.
David J. Linden;Michael H. Dickinson;Michelle Smeyne;John A. Connor.
Neuron (1991)
Global Ischemia Induces Downregulation of Glur2 mRNA and Increases AMPA Receptor-Mediated Ca2+ Influx in Hippocampal CA1 Neurons of Gerbil
J.A. Gorter;J.J. Petrozzino;E.M.A. Aronica;D.M. Rosenbaum.
The Journal of Neuroscience (1997)
Physiological and structural evidence for hippocampal involvement in persistent seizure susceptibility after traumatic brain injury.
Golijeh Golarai;Anders C. Greenwood;Dennis M. Feeney;John A. Connor.
The Journal of Neuroscience (2001)
Electrical activity in cerebellar cultures determines Purkinje cell dendritic growth patterns
Karl Schilling;Michael H. Dickinson;John A. Connor;James I. Morgan.
Neuron (1991)
Cellular mechanisms of long-term depression in the cerebellum
David J. Linden;John A. Connor.
Current Opinion in Neurobiology (1993)
Bidirectional synaptic plasticity correlated with the magnitude of dendritic calcium transients above a threshold.
R. J. Cormier;A. C. Greenwood;J. A. Connor.
Journal of Neurophysiology (2001)
NAD(P)H fluorescence imaging of postsynaptic neuronal activation in murine hippocampal slices.
C. William Shuttleworth;Angela M. Brennan;John A. Connor.
The Journal of Neuroscience (2003)
Long-term Depression of Glutamate Currents in Cultured Cerebellar Purkinje Neurons Does Not Require Nitric Oxide Signalling.
David J. Linden;John A. Connor.
European Journal of Neuroscience (1992)
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