D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 31 Citations 5,554 53 World Ranking 6983 National Ranking 2935

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • Hippocampus

His primary scientific interests are in Neuroscience, Hippocampal formation, Hippocampus, Corticotropin-releasing hormone and Synaptic plasticity. His Chronic stress, Cognition and Neocortex study in the realm of Neuroscience interacts with subjects such as Corticotropin-releasing hormone receptor 1 and Monoaminergic cell groups. His work deals with themes such as Hyperpolarization, Depolarization, Transcriptional regulation, Premovement neuronal activity and Gene isoform, which intersect with Hippocampal formation.

His study in Hippocampus is interdisciplinary in nature, drawing from both Dendritic spine and Long-term potentiation. His work in Corticotropin-releasing hormone addresses subjects such as Neuropeptide, which are connected to disciplines such as In vitro, Endogeny and Hippocampus. His studies in Synaptic plasticity integrate themes in fields like Mutation, Chromatin, Chromatin remodeling and Long-term memory.

His most cited work include:

  • Mechanisms of Late-Onset Cognitive Decline after Early-Life Stress (338 citations)
  • Hippocampal Dysfunction and Cognitive Impairments Provoked by Chronic Early-Life Stress Involve Excessive Activation of CRH Receptors (269 citations)
  • Developmental Febrile Seizures Modulate Hippocampal Gene Expression of Hyperpolarization-Activated Channels in an Isoform- and Cell-Specific Manner (237 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Neuroscience, Hippocampal formation, Hippocampus, Corticotropin-releasing hormone and Dendritic spine. His research on Neuroscience frequently links to adjacent areas such as Synaptic plasticity. His work on Pyramidal cell as part of general Hippocampal formation research is frequently linked to Population, bridging the gap between disciplines.

The Hippocampus study combines topics in areas such as Glutamate receptor, Long-term potentiation, Neurochemical and Inhibitory postsynaptic potential. His Corticotropin-releasing hormone research integrates issues from Neuropeptide and Stria terminalis. His study looks at the relationship between Dendritic spine and fields such as Synapse, as well as how they intersect with chemical problems.

He most often published in these fields:

  • Neuroscience (73.58%)
  • Hippocampal formation (47.17%)
  • Hippocampus (41.51%)

What were the highlights of his more recent work (between 2017-2021)?

  • Neuroscience (73.58%)
  • Amygdala (13.21%)
  • Hippocampus (41.51%)

In recent papers he was focusing on the following fields of study:

Yuncai Chen mostly deals with Neuroscience, Amygdala, Hippocampus, Hippocampal formation and Anxiety. Yuncai Chen combines subjects such as Transcription factor and Epigenetics with his study of Neuroscience. His Amygdala research includes elements of Effects of stress on memory, Nucleus accumbens, Chronic stress and Corticotropin-releasing hormone.

His Corticotropin-releasing hormone study combines topics in areas such as Neuropeptide, Stria terminalis and Basolateral amygdala. His research in Hippocampus intersects with topics in Inhibitory postsynaptic potential, Neurochemical and Parvalbumin. Yuncai Chen merges many fields, such as Hippocampal formation and Population, in his writings.

Between 2017 and 2021, his most popular works were:

  • Anhedonia Following Early-Life Adversity Involves Aberrant Interaction of Reward and Anxiety Circuits and Is Reversed by Partial Silencing of Amygdala Corticotropin-Releasing Hormone Gene. (68 citations)
  • NRSF-dependent epigenetic mechanisms contribute to programming of stress-sensitive neurons by neonatal experience, promoting resilience. (54 citations)
  • Plasticity of the Reward Circuitry After Early-Life Adversity: Mechanisms and Significance. (20 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • Hippocampus

Yuncai Chen mainly investigates Neuroscience, Amygdala, Anxiety, Anhedonia and Pleasure. His Neuroscience study frequently links to other fields, such as Epigenetics. His Epigenetics research incorporates themes from Neurotransmission, Neuron, Chromatin, Repressor and Gene silencing.

His work carried out in the field of Gene silencing brings together such families of science as Glutamatergic and Transcription factor. His biological study spans a wide range of topics, including Nucleus accumbens and Addiction. Yuncai Chen has included themes like Schizophrenia, Nerve net, Corticotropin-releasing hormone, Central nucleus of the amygdala and Developmental psychology in his Prefrontal cortex study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mechanisms of Late-Onset Cognitive Decline after Early-Life Stress

Kristen L. Brunson;Enikö Kramár;Bin Lin;Yuncai Chen.
The Journal of Neuroscience (2005)

478 Citations

Hippocampal Dysfunction and Cognitive Impairments Provoked by Chronic Early-Life Stress Involve Excessive Activation of CRH Receptors

Autumn S. Ivy;Christopher S. Rex;Yuncai Chen;Céline Dubé.
The Journal of Neuroscience (2010)

398 Citations

Toward Understanding How Early-Life Stress Reprograms Cognitive and Emotional Brain Networks.

Yuncai Chen;Tallie Z Baram.
Neuropsychopharmacology (2016)

348 Citations

Developmental Febrile Seizures Modulate Hippocampal Gene Expression of Hyperpolarization-Activated Channels in an Isoform- and Cell-Specific Manner

Amy Brewster;Roland A. Bender;Yuncai Chen;Celine Dube.
The Journal of Neuroscience (2002)

310 Citations

Immunocytochemical Distribution of Corticotropin-Releasing Hormone Receptor Type-1 (CRF1)-Like Immunoreactivity in the Mouse Brain: Light Microscopy Analysis Using an Antibody Directed Against the C-Terminus

Yuncai Chen;Kristen L. Brunson;Marianne B. Müller;Wayna Cariaga.
The Journal of Comparative Neurology (2000)

296 Citations

Rapid Loss of Dendritic Spines after Stress Involves Derangement of Spine Dynamics by Corticotropin-Releasing Hormone

Yuncai Chen;Céline M. Dubé;Courtney J. Rice;Tallie Z. Baram.
The Journal of Neuroscience (2008)

280 Citations

Correlated memory defects and hippocampal dendritic spine loss after acute stress involve corticotropin-releasing hormone signaling

Yuncai Chen;Christopher S. Rex;Courtney J. Rice;Céline M. Dubé.
Proceedings of the National Academy of Sciences of the United States of America (2010)

273 Citations

Long-term, progressive hippocampal cell loss and dysfunction induced by early-life administration of corticotropin-releasing hormone reproduce the effects of early-life stress

Kristen L. Brunson;Mariam Eghbal-Ahmadi;Roland Bender;Yuncai Chen.
Proceedings of the National Academy of Sciences of the United States of America (2001)

272 Citations

The neuron-specific chromatin regulatory subunit BAF53b is necessary for synaptic plasticity and memory.

Annie Vogel-Ciernia;Dina P. Matheos;Ruth M. Barrett;Enikoe A. Kramar.
Nature Neuroscience (2013)

236 Citations

Modulation of dendritic differentiation by corticotropin-releasing factor in the developing hippocampus.

Yuncai Chen;Roland A. Bender;Kristen L. Brunson;Jörn K. Pomper.
Proceedings of the National Academy of Sciences of the United States of America (2004)

235 Citations

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