His scientific interests lie mostly in Cell biology, Reactive oxygen species, Apoptosis, Inflammasome and Pharmacology. His Cell biology research is multidisciplinary, relying on both Caspase, Immune system and Superoxide. His Reactive oxygen species research is multidisciplinary, incorporating perspectives in Superoxide dismutase, Photochemistry, Phosphorylation, Spin trapping and Programmed cell death.
His work in the fields of Apoptosis, such as Fas ligand, overlaps with other areas such as Dephosphorylation. Yon Rojanasakul interconnects Innate immune system and Signal transducing adaptor protein in the investigation of issues within Inflammasome. His Pharmacology research is multidisciplinary, incorporating elements of Cancer and Cytotoxicity.
Yon Rojanasakul spends much of his time researching Cancer research, Cell biology, Apoptosis, Cancer and Reactive oxygen species. His work carried out in the field of Cancer research brings together such families of science as Cell, Protein kinase B, Cell growth, Carcinogenesis and Lung cancer. His study in Carcinogenesis is interdisciplinary in nature, drawing from both Malignant transformation, Immunology and Carcinogen.
His study focuses on the intersection of Cell biology and fields such as S-Nitrosylation with connections in the field of Nitric oxide. His research integrates issues of Cancer cell and Superoxide in his study of Apoptosis. His work deals with themes such as Oxidative stress, Superoxide dismutase, Catalase and Biophysics, which intersect with Reactive oxygen species.
Yon Rojanasakul mostly deals with Cancer research, Lung cancer, Nanotechnology, Cancer and Cell biology. His Cancer research research includes elements of Apoptosis, Protein kinase B, Cell cycle, Cisplatin and Ovarian cancer. His research in Lung cancer intersects with topics in Cell, Metastasis and Motility.
His work on Drug delivery as part of general Nanotechnology research is frequently linked to Food packaging, bridging the gap between disciplines. The various areas that he examines in his Cell biology study include Downregulation and upregulation, In vivo and Transcription factor. His research investigates the connection between Signal transduction and topics such as Viability assay that intersect with issues in Reactive oxygen species.
His primary areas of study are Nanotechnology, Apoptosis, Cancer research, Cell cycle and Cell biology. Yon Rojanasakul combines subjects such as Human lung and Polymer with his study of Nanotechnology. His research on Apoptosis often connects related areas such as Cell growth.
His Cancer research research incorporates elements of Cancer, Ovarian cancer, Protein kinase B, G1 phase and Cyclin E. Yon Rojanasakul regularly links together related areas like Transcription factor in his Cell biology studies. His work in Drug delivery tackles topics such as Genotoxicity which are related to areas like Oxidative stress.
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Mechanisms of nanoparticle-induced oxidative stress and toxicity.
Amruta Manke;Liying Wang;Yon Rojanasakul.
BioMed Research International (2013)
Inflammation and Lung Cancer: Roles of Reactive Oxygen/Nitrogen Species
Neelam Azad;Yon Rojanasakul;Val Vallyathan.
Journal of Toxicology and Environmental Health-part B-critical Reviews (2008)
Protein Nanoparticles as Drug Delivery Carriers for Cancer Therapy
Warangkana Lohcharoenkal;Liying Wang;Yi Charlie Chen;Yon Rojanasakul.
BioMed Research International (2014)
Oxygen radical-mediated pulmonary toxicity induced by some cationic liposomes.
Sujatha Dokka;David Toledo;Xianglin Shi;Vincent Castranova.
Pharmaceutical Research (2000)
An NLRP7-containing inflammasome mediates recognition of microbial lipopeptides in human macrophages.
Sonal Khare;Andrea Dorfleutner;Nicole B. Bryan;Nicole B. Bryan;Chawon Yun.
Immunity (2012)
Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: The challenge ahead
William H Goodson;Leroy Lowe;David O Carpenter;Michael Gilbertson.
Carcinogenesis (2015)
Role of PI3K and AKT specific isoforms in ovarian cancer cell migration, invasion and proliferation through the p70S6K1 pathway
Qiao Meng;Chang Xia;Jing Fang;Yon Rojanasakul.
Cellular Signalling (2006)
Activation of inflammasomes requires intracellular redistribution of the apoptotic speck-like protein containing a caspase recruitment domain.
Nicole B. Bryan;Andrea Dorfleutner;Yon Rojanasakul;Christian Stehlik.
Journal of Immunology (2009)
Reactive Oxygen Species Mediate Caspase Activation and Apoptosis Induced by Lipoic Acid in Human Lung Epithelial Cancer Cells through Bcl-2 Down-Regulation
Jirapan Moungjaroen;Ubonthip Nimmannit;Patrick S Callery;Liying Wang.
Journal of Pharmacology and Experimental Therapeutics (2006)
S-Nitrosylation of Bcl-2 Inhibits Its Ubiquitin-Proteasomal Degradation: A NOVEL ANTIAPOPTOTIC MECHANISM THAT SUPPRESSES APOPTOSIS *
Neelam Azad;Val Vallyathan;Liying Wang;Vimon Tantishaiyakul.
Journal of Biological Chemistry (2006)
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