2000 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of study are Cell biology, Biochemistry, Endosome, Endocytosis and Receptor. His research integrates issues of Type three secretion system, Salmonella and Membrane ruffling in his study of Cell biology. His Biochemistry study frequently intersects with other fields, such as Molecular biology.
His biological study spans a wide range of topics, including Vesicle, Endocytic cycle, ADP ribosylation factor and Lipid bilayer fusion. Philip D. Stahl has researched Endocytosis in several fields, including Guanine, Transferrin, Signal transduction and Intracellular. His work on Internalization and Receptor recycling as part of general Receptor study is frequently connected to Marginal zone, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Philip D. Stahl spends much of his time researching Cell biology, Endosome, Biochemistry, Endocytosis and Endocytic cycle. His research in Cell biology focuses on subjects like GTP', which are connected to GTP-binding protein regulators. His Endosome research includes elements of Lipid bilayer fusion, Lysosome, Cathepsin D, Vesicle and Endocytic vesicle.
His Biochemistry and Mannose, Mannose receptor, Receptor, Glycoprotein and Ligand investigations all form part of his Biochemistry research activities. His Endocytosis study incorporates themes from Molecular biology, Internalization, Intracellular and Cytosol. His study in the field of Bulk endocytosis also crosses realms of Vesicle fusion.
Philip D. Stahl mainly focuses on Cell biology, Extracellular vesicles, Endosome, Phosphorylation and Rab. His Cell biology research incorporates elements of Endocytic cycle, Endocytosis and Insulin. Many of his studies on Endocytosis involve topics that are commonly interrelated, such as Mannose receptor.
His work carried out in the field of Extracellular vesicles brings together such families of science as Neuroscience and Homeostasis. His Endosome study is related to the wider topic of Cell. His research in Rab intersects with topics in Fucose, Glucuronidase, Glycoconjugate and Bovine serum albumin.
Philip D. Stahl mostly deals with Cell biology, Extracellular vesicles, Extracellular vesicle, Endosome and CD36. His Phosphorylation, AMPK, Rab, Transport protein and Intracellular study are his primary interests in Cell biology. His Rab study combines topics in areas such as Transferrin receptor, Transferrin transport, Brefeldin A, Endocytic cycle and Internalization.
Philip D. Stahl has included themes like Neuroscience and Bioinformatics in his Extracellular vesicles study. His Endosome research integrates issues from Transferrin, Cell signaling, Signal transduction, Homeostasis and Membrane. His CD36 research is multidisciplinary, incorporating elements of Lysine, Oleic acid and adipocyte protein 2, Fatty acid.
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Minimal information for studies of extracellular vesicles 2018 (MISEV2018) : a position statement of the International Society for Extracellular Vesicles and update of the MISEV2014 guidelines
Clotilde Théry;Kenneth W. Witwer;Elena Aikawa;Maria Jose Alcaraz.
Journal of extracellular vesicles (2018)
Receptor-mediated endocytosis of transferrin and recycling of the transferrin receptor in rat reticulocytes
C Harding;J Heuser;P Stahl.
Journal of Cell Biology (1983)
The mannose receptor is a pattern recognition receptor involved in host defense.
Philip D Stahl;R Alan B Ezekowitz.
Current Opinion in Immunology (1998)
Evidence for receptor-mediated binding of glycoproteins, glycoconjugates, and lysosomal glycosidases by alveolar macrophages.
P. D. Stahl;J. S. Rodman;M. J. Miller;Paul Schlesinger.
Proceedings of the National Academy of Sciences of the United States of America (1978)
Receptor-mediated pinocytosis of mannose glycoconjugates by macrophages: Characterization and evidence for receptor recycling
Philip Stahl;Paul H. Schlesinger;Elaine Sigardson;Jane S. Rodman.
A Salmonella virulence protein that inhibits cellular trafficking.
Kei‐ichi Uchiya;M.Alejandro Barbieri;Kouichi Funato;Ankur H. Shah.
The EMBO Journal (1999)
T. Wileman;Clifford V Harding;P. Stahl.
Biochemical Journal (1985)
A regulatory role for ARF6 in receptor-mediated endocytosis.
Crislyn D'Souza-Schorey;Guangpu Li;Maria I. Colombo;Philip D. Stahl.
Mannose Receptor and Its Putative Ligands in Normal Murine Lymphoid and Nonlymphoid Organs: In Situ Expression of Mannose Receptor by Selected Macrophages, Endothelial Cells, Perivascular Microglia, and Mesangial Cells, but not Dendritic Cells
Sheena A. Linehan;Luisa Martínez-Pomares;Philip D. Stahl;Siamon Gordon.
Journal of Experimental Medicine (1999)
Modulation of Rab5 and Rab7 recruitment to phagosomes by phosphatidylinositol 3-kinase.
Otilia V. Vieira;Cecilia Bucci;Rene E. Harrison;William S. Trimble.
Molecular and Cellular Biology (2003)
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