Jesús Pla mainly focuses on Candida albicans, Mitogen-activated protein kinase, Corpus albicans, Kinase and Gene. Candida albicans and Mutant are commonly linked in his work. His Mitogen-activated protein kinase study falls within the topics of Cell biology and Biochemistry.
His Corpus albicans study deals with Saccharomyces cerevisiae intersecting with Complementation. His study in the fields of MAPK14 and MAPK7 under the domain of Kinase overlaps with other disciplines such as MAPK1. His Gene research incorporates elements of Organism and Molecular biology.
His primary scientific interests are in Candida albicans, Microbiology, Cell biology, Corpus albicans and Biochemistry. His study in Candida albicans is interdisciplinary in nature, drawing from both Mutant, Gene, Saccharomyces cerevisiae, Fungal protein and Mitogen-activated protein kinase. His Mitogen-activated protein kinase research is within the category of Kinase.
His Microbiology research integrates issues from In vitro, Cell wall, Immune system and Virulence. The various areas that Jesús Pla examines in his Cell biology study include Oxidative stress and Biogenesis. His studies deal with areas such as Adaptation, Wild type and Kinase activity as well as Corpus albicans.
The scientist’s investigation covers issues in Candida albicans, Corpus albicans, Microbiology, Cell biology and Gene. His Candida albicans study incorporates themes from Epithelium, Transcription factor, Osmotic shock, Computational biology and MAPK/ERK pathway. The Corpus albicans study combines topics in areas such as Pathogen, Hypha, Pathogenic fungus and Virulence.
His Microbiology research focuses on Immune system and how it relates to Receptor and Mutation. His biological study spans a wide range of topics, including Psychological repression, Regulation of gene expression, Transcriptome and Activator. In his research, Molecular genetics is intimately related to Cell, which falls under the overarching field of Gene.
Cell biology, Gene, Candida albicans, Transcription factor and MAPK/ERK pathway are his primary areas of study. The various areas that Jesús Pla examines in his Cell biology study include RNA, Repressor, Cas9, Activator and Regulation of gene expression. Jesús Pla interconnects Receptor, Immunity and Microbiology in the investigation of issues within Gene.
His studies deal with areas such as Tetracycline, Epigenetics and Saccharomyces cerevisiae as well as Candida albicans. His study in MAPK/ERK pathway is interdisciplinary in nature, drawing from both CHEK1, Cell cycle checkpoint, Mitogen-activated protein kinase and Wild type. His Mitogen-activated protein kinase research is multidisciplinary, incorporating perspectives in Cell cycle and Cyclin.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Role of the Mitogen-Activated Protein Kinase Hog1p in Morphogenesis and Virulence of Candida albicans
R. Alonso-Monge;F. Navarro-García;G. Molero;R. Diez-Orejas.
Journal of Bacteriology (1999)
The Hog1 mitogen-activated protein kinase is essential in the oxidative stress response and chlamydospore formation in Candida albicans.
Rebeca Alonso-Monge;Federico Navarro-García;Elvira Román;Ana I. Negredo.
Eukaryotic Cell (2003)
The MAP kinase signal transduction network in Candida albicans
R. Alonso Monge;E. Román;C. Nombela;J. Pla.
Microbiology (2006)
Virulence genes in the pathogenic yeast Candida albicans.
Federico Navarro-García;Miguel Sánchez;César Nombela;Jesús Pla.
Fems Microbiology Reviews (2001)
Division genes in Escherichia coli are expressed coordinately to cell septum requirements by gearbox promoters.
M Aldea;T Garrido;J Pla;M Vicente.
The EMBO Journal (1990)
Functional characterization of the MKC1 gene of Candida albicans, which encodes a mitogen-activated protein kinase homolog related to cell integrity.
F Navarro-García;M Sánchez;J Pla;C Nombela.
Molecular and Cellular Biology (1995)
Candida albicans: genetics, dimorphism and pathogenicity.
Gloria Molero;Rosalía Díez-Orejas;Federico Navarro-García;Lucía Monteoliva.
International Microbiology (1998)
The mitogen-activated protein kinase homolog HOG1 gene controls glycerol accumulation in the pathogenic fungus Candida albicans.
C San José;R A Monge;R Pérez-Díaz;J Pla.
Journal of Bacteriology (1996)
The Sho1 adaptor protein links oxidative stress to morphogenesis and cell wall biosynthesis in the fungal pathogen Candida albicans.
Elvira Román;César Nombela;Jesús Pla.
Molecular and Cellular Biology (2005)
MAP kinase pathways as regulators of fungal virulence
Elvira Román;David M. Arana;César Nombela;Rebeca Alonso-Monge.
Trends in Microbiology (2007)
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