What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Cancer
His main research concerns Internal medicine, Immunology, Inflammation, Endocrinology and Proinflammatory cytokine.
His research is interdisciplinary, bridging the disciplines of Gastroenterology and Internal medicine.
Immunology is often connected to Inflammatory bowel disease in his work.
His research in Inflammation intersects with topics in Haematopoiesis, Osteoprotegerin, Bioinformatics, Immunity and Pharmacology.
His research in the fields of Adipose tissue and Adiponectin overlaps with other disciplines such as Thrombopoiesis.
His Proinflammatory cytokine research incorporates themes from Inflammasome, Interferon gamma, Neopterin, Phytohaemagglutinin and Interleukin.
His most cited work include:
- Adipocytokines: mediators linking adipose tissue, inflammation and immunity. (2263 citations)
- EASL-EASD-EASO Clinical Practice Guidelines for the Management of Non-Alcoholic Fatty Liver Disease (1673 citations)
- Evolution of inflammation in nonalcoholic fatty liver disease: The multiple parallel hits hypothesis† (1347 citations)
What are the main themes of his work throughout his whole career to date?
Herbert Tilg focuses on Internal medicine, Immunology, Endocrinology, Gastroenterology and Inflammation.
Internal medicine is represented through his Fatty liver, Disease, Insulin resistance, Cirrhosis and Ulcerative colitis research.
His Fatty liver research is multidisciplinary, relying on both Fibrosis, Steatosis and Diabetes mellitus.
His Ulcerative colitis study combines topics in areas such as Crohn's disease and Inflammatory bowel disease.
His study involves Cytokine, Immune system, Tumor necrosis factor alpha, Proinflammatory cytokine and Gut flora, a branch of Immunology.
Adipose tissue, Adiponectin, Adipokine, Weight loss and Metabolic syndrome are the subjects of his Endocrinology studies.
He most often published in these fields:
- Internal medicine (48.59%)
- Immunology (27.11%)
- Endocrinology (22.49%)
What were the highlights of his more recent work (between 2017-2021)?
- Internal medicine (48.59%)
- Disease (14.66%)
- Gastroenterology (19.88%)
In recent papers he was focusing on the following fields of study:
Herbert Tilg mainly investigates Internal medicine, Disease, Gastroenterology, Fatty liver and Endocrinology.
Herbert Tilg interconnects Microbiome, Biopsy, Clinical trial and Gut flora in the investigation of issues within Disease.
Herbert Tilg combines subjects such as Diabetes mellitus and Insulin resistance with his study of Fatty liver.
His Endocrinology study focuses on Adipose tissue in particular.
Herbert Tilg works mostly in the field of Liver transplantation, limiting it down to topics relating to Liver disease and, in certain cases, Inflammation, as a part of the same area of interest.
Herbert Tilg frequently studies issues relating to Immunology and Cirrhosis.
Between 2017 and 2021, his most popular works were:
- The Intestinal Microbiota in Colorectal Cancer (203 citations)
- Recovery of ethanol-induced Akkermansia muciniphila depletion ameliorates alcoholic liver disease. (188 citations)
- The intestinal microbiota fuelling metabolic inflammation (148 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Cancer
Herbert Tilg spends much of his time researching Internal medicine, Fatty liver, Disease, Immunology and Meta-analysis.
His Internal medicine study integrates concerns from other disciplines, such as Gastroenterology, Endocrinology and Pathology.
The various areas that Herbert Tilg examines in his Fatty liver study include Diabetes mellitus, Cirrhosis and Phase iii trials.
Herbert Tilg has researched Disease in several fields, including Microbiome, Cardiomyopathy, Pathophysiology and Kidney disease.
His Inflammation, Cytokine, Proinflammatory cytokine, Interleukin 17 and Interleukin 23 investigations are all subjects of Immunology research.
He usually deals with Insulin resistance and limits it to topics linked to Interferon and Immune system.
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