Emanuela Corsini spends much of her time researching Immunology, Immune system, Cell biology, Cytokine and Biochemistry. Immunology is closely attributed to Pathology in her study. Her Immune system research includes elements of Epidemiology, In vitro toxicology, Toxicity and Immunopathology.
Her Cell biology research incorporates themes from Receptor, Neurodegeneration and Programmed cell death. The study incorporates disciplines such as Tumor necrosis factor alpha, Progenitor cell, Signal transduction and Nervous system in addition to Cytokine. Emanuela Corsini is interested in In vitro, which is a branch of Biochemistry.
Her primary areas of study are Immunology, Immune system, In vitro, Pharmacology and Cytokine. Her Immune system study incorporates themes from Toxicity and Immunopathology. Her work deals with themes such as Molecular biology and Interleukin 8, which intersect with In vitro.
Emanuela Corsini combines subjects such as Skin sensitization, Potency, In vivo and Mechanism of action with her study of Pharmacology. Her research in Cytokine intersects with topics in Inflammation and Signal transduction. Her Tumor necrosis factor alpha research also covers Endocrinology and Internal medicine studies.
Emanuela Corsini mostly deals with Immunology, Immune system, Cell biology, In vitro and Receptor for activated C kinase 1. Her research investigates the connection between Immunology and topics such as Drug that intersect with issues in Adverse effect and Allergy. She interconnects Hormone, Endocrine system and Disease in the investigation of issues within Immune system.
Her biological study deals with issues like Pharmacology, which deal with fields such as Skin sensitization, Cell activation and Carcinogen. Her Receptor for activated C kinase 1 study incorporates themes from Endocrinology, Glucocorticoid receptor and Internal medicine. Her Endocrinology research focuses on Cytokine and how it connects with Whole blood, Tumor necrosis factor alpha and Innate immune system.
Immunology, Immune system, Receptor for activated C kinase 1, Cytokine and Cell biology are her primary areas of study. Her Immunology research integrates issues from Endogeny, Lung and Homeostasis. She interconnects Cell activation, Disease and Pharmacology in the investigation of issues within Immune system.
Her Receptor for activated C kinase 1 study combines topics from a wide range of disciplines, such as Internal medicine, Glucocorticoid receptor and Endocrinology. Her Cytokine research includes themes of Bronchoalveolar lavage, Recurrent airway obstruction, Mesenchymal stem cell and Stem cell. Her study explores the link between Cell biology and topics such as Transcriptional regulation that cross with problems in Kinase, Mediator and Scaffold protein.
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Interleukin-1β Enhances NMDA Receptor-Mediated Intracellular Calcium Increase through Activation of the Src Family of Kinases
Barbara Viviani;S. Bartesaghi;F. Gardoni;A. Vezzani.
The Journal of Neuroscience (2003)
Risk Assessment in Immunotoxicology: II. Relationships between Immune and Host Resistance Tests
Michael I. Luster;Christopher Portier;D. Gayla Pait;Gary J. Rosenthal.
Toxicological Sciences (1993)
Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: The challenge ahead
William H Goodson;Leroy Lowe;David O Carpenter;Michael Gilbertson.
Immune response in COVID-19: addressing a pharmacological challenge by targeting pathways triggered by SARS-CoV-2.
Michele Catanzaro;Francesca Fagiani;Francesca Fagiani;Marco Racchi;Emanuela Corsini.
Signal Transduction and Targeted Therapy (2020)
Pesticide induced immunotoxicity in humans: A comprehensive review of the existing evidence
E. Corsini;M. Sokooti;C.L. Galli;A. Moretto.
Erythropoietin protects primary hippocampal neurons increasing the expression of brain-derived neurotrophic factor.
Barbara Viviani;Stefano Bartesaghi;Emanuela Corsini;Pia Villa.
Journal of Neurochemistry (2005)
Cytokines and irritant contact dermatitis.
Emanuela Corsini;Corrado L Galli.
Toxicology Letters (1998)
Organotins induce apoptosis by disturbance of [Ca2+]i and mitochondrial activity, causing oxidative stress and activation of caspases in rat thymocytes
Alessandra Gennari;Alessandra Gennari;Barbara Viviani;Corrado Lodovico Galli;Marina Marinovich.
Toxicology and Applied Pharmacology (2000)
Epidermal cytokines in experimental contact dermatitis.
Emanuela Corsini;Corrado L Galli.
Glia increase degeneration of hippocampal neurons through release of tumor necrosis factor-α
Barbara Viviani;Emanuela Corsini;Corrado L. Galli;Marina Marinovich.
Toxicology and Applied Pharmacology (1998)
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