D-Index & Metrics Best Publications
Craig A. Stockmeier

Craig A. Stockmeier

University of Mississippi Medical Center
United States

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Dopamine
  • Neurotransmitter

His primary scientific interests are in Neuroscience, Prefrontal cortex, Major depressive disorder, Internal medicine and Endocrinology. His Neuroscience study frequently intersects with other fields, such as Glutamatergic. His work on Dorsolateral prefrontal cortex as part of general Prefrontal cortex study is frequently connected to Eukaryotic initiation factor, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.

His research in Major depressive disorder intersects with topics in mTORC1, Chronic stress and Molecular neuroscience. His Internal medicine research is multidisciplinary, incorporating perspectives in Anesthesia and Typical antipsychotic, Antipsychotic, Atypical antipsychotic. The concepts of his Endocrinology study are interwoven with issues in Clozapine, Serotonergic, Pharmacology and In vivo.

His most cited work include:

  • Morphometric evidence for neuronal and glial prefrontal cell pathology in major depression (1234 citations)
  • Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression (1131 citations)
  • Cellular changes in the postmortem hippocampus in major depression. (553 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Internal medicine, Endocrinology, Neuroscience, Prefrontal cortex and Major depressive disorder. Internal medicine is closely attributed to Anesthesia in his work. His studies in Endocrinology integrate themes in fields like Receptor and Serotonin.

His research links Glutamate receptor with Neuroscience. The study incorporates disciplines such as GABAergic, Astrocyte and Pathology in addition to Prefrontal cortex. His Major depressive disorder research is multidisciplinary, incorporating elements of Dentate gyrus, Postmortem studies and Bioinformatics.

He most often published in these fields:

  • Internal medicine (68.31%)
  • Endocrinology (67.90%)
  • Neuroscience (53.91%)

What were the highlights of his more recent work (between 2016-2020)?

  • Internal medicine (68.31%)
  • Depression (16.46%)
  • Major depressive disorder (49.38%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Internal medicine, Depression, Major depressive disorder, Endocrinology and Psychiatry. Craig A. Stockmeier combines subjects such as Bioinformatics, Prefrontal cortex, Axon, Messenger RNA and Genetic architecture with his study of Major depressive disorder. His Bioinformatics research includes elements of Dentate gyrus, Hippocampus, Hippocampal formation, Transcriptome and Neurogenesis.

His work carried out in the field of Prefrontal cortex brings together such families of science as Chromatin, Cause of death and Genetic association. His research integrates issues of Neuroscience, Dorsolateral prefrontal cortex, Neurotransmission and Serotonin in his study of Endocrinology. As part of his studies on Neuroscience, Craig A. Stockmeier frequently links adjacent subjects like Psychosis.

Between 2016 and 2020, his most popular works were:

  • Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression (1131 citations)
  • Evaluation of chromatin accessibility in prefrontal cortex of individuals with schizophrenia (57 citations)
  • Altered neuro-inflammatory gene expression in hippocampus in major depressive disorder. (54 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Dopamine
  • Neurotransmitter

His primary areas of study are Major depressive disorder, Axon, Gene expression, Prefrontal cortex and Bioinformatics. His Major depressive disorder study introduces a deeper knowledge of Endocrinology. His Axon study is associated with Neuroscience.

His Gene expression research incorporates themes from Hippocampal formation, Neurogenesis and Hippocampus. His study looks at the relationship between Prefrontal cortex and topics such as Soma, which overlap with Pathology, Myelin and Messenger RNA. Craig A. Stockmeier has researched Bioinformatics in several fields, including Dentate gyrus, Meta-analysis, Depression and Genetic architecture.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Morphometric evidence for neuronal and glial prefrontal cell pathology in major depression

Grazyna Rajkowska;José J Miguel-Hidalgo;Jinrong Wei;Ginny Dilley.
Biological Psychiatry (1999)

1806 Citations

Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression

Naomi R. Wray;Stephan Ripke;Stephan Ripke;Stephan Ripke;Manuel Mattheisen;MacIej Trzaskowski.
Nature Genetics (2018)

1759 Citations

Cellular changes in the postmortem hippocampus in major depression.

Craig A. Stockmeier;Craig A. Stockmeier;Gouri J. Mahajan;Lisa C. Konick;James C. Overholser.
Biological Psychiatry (2004)

787 Citations

Decreased expression of synapse-related genes and loss of synapses in major depressive disorder

Hyo Jung Kang;Bhavya Voleti;Tibor Hajszan;Tibor Hajszan;Grazyna Rajkowska.
Nature Medicine (2012)

638 Citations

Astrocyte Pathology in Major Depressive Disorder: Insights from Human Postmortem Brain Tissue

Grazyna Rajkowska;Craig A Stockmeier.
Current Drug Targets (2013)

552 Citations

Increase in serotonin-1A autoreceptors in the midbrain of suicide victims with major depression-postmortem evidence for decreased serotonin activity.

Craig A. Stockmeier;Laura A. Shapiro;Ginny E. Dilley;Tamara N. Kolli.
The Journal of Neuroscience (1998)

543 Citations

Reduced Levels of Norepinephrine Transporters in the Locus Coeruleus in Major Depression

Violetta Klimek;Craig Stockmeier;James Overholser;Herbert Y. Meltzer.
The Journal of Neuroscience (1997)

504 Citations

Increased nicotinic receptors in brains from smokers: membrane binding and autoradiography studies.

David C. Perry;Martha I. Dávila-Garcı́a;Craig A. Stockmeier;Kenneth J. Kellar.
Journal of Pharmacology and Experimental Therapeutics (1999)

459 Citations

GABAergic neurons immunoreactive for calcium binding proteins are reduced in the prefrontal cortex in major depression.

Grazyna Rajkowska;Gillian O'Dwyer;Zsofia Teleki;Craig A Stockmeier;Craig A Stockmeier.
Neuropsychopharmacology (2007)

423 Citations

A negative regulator of MAP kinase causes depressive behavior

Vanja Duric;Mounira Banasr;Pawel Licznerski;Heath D Schmidt.
Nature Medicine (2010)

385 Citations

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