D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 31 Citations 3,951 64 World Ranking 5103 National Ranking 2202

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Enzyme
  • Biochemistry

Hippocampal formation, Neuroscience, Ischemia, Ischemic preconditioning and Internal medicine are his primary areas of study. The Hippocampal formation study which covers Hippocampus that intersects with Anesthesia, Synaptic plasticity, Tetanic stimulation, Long-term potentiation and Excitatory postsynaptic potential. Thomas J. Sick combines subjects such as Turtle and Depolarization with his study of Neuroscience.

His work deals with themes such as Mitochondrion, Programmed cell death and Pathology, which intersect with Ischemia. As a member of one scientific family, he mostly works in the field of Ischemic preconditioning, focusing on Neuroprotection and, on occasion, Signal transduction and Protein kinase C. The Internal medicine study combines topics in areas such as Endocrinology and Oxidative phosphorylation.

His most cited work include:

  • Increased seizure duration and slowed potassium kinetics in mice lacking aquaporin-4 water channels. (274 citations)
  • Rapid preconditioning protects rats against ischemic neuronal damage after 3 but not 7 days of reperfusion following global cerebral ischemia (202 citations)
  • εPKC Is Required for the Induction of Tolerance by Ischemic and NMDA-Mediated Preconditioning in the Organotypic Hippocampal Slice (200 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Neuroscience, Internal medicine, Endocrinology, Ischemia and Hippocampal formation. His Neuroscience study incorporates themes from Synaptic plasticity, Biophysics, Depolarization and Neurotransmission. While the research belongs to areas of Internal medicine, Thomas J. Sick spends his time largely on the problem of Anesthesia, intersecting his research to questions surrounding Status epilepticus and Epilepsy.

His biological study spans a wide range of topics, including Apoptosis, Cytochrome c and Nicotine. His Ischemia research incorporates themes from Cerebral cortex, Programmed cell death and Mitochondrion. Long-term potentiation is closely connected to Hippocampus in his research, which is encompassed under the umbrella topic of Hippocampal formation.

He most often published in these fields:

  • Neuroscience (30.28%)
  • Internal medicine (31.19%)
  • Endocrinology (27.52%)

What were the highlights of his more recent work (between 2006-2021)?

  • Neuroscience (30.28%)
  • Long-term potentiation (6.42%)
  • Hippocampus (13.76%)

In recent papers he was focusing on the following fields of study:

Thomas J. Sick mainly investigates Neuroscience, Long-term potentiation, Hippocampus, Synaptic plasticity and Epilepsy. Thomas J. Sick has included themes like Hyperthermia and Metabolism in his Neuroscience study. Thomas J. Sick has researched Long-term potentiation in several fields, including Hippocampal formation, Neuroprotection, Nicotine and Nicotinamide phosphoribosyltransferase.

His research on Hippocampal formation is centered around Endocrinology and Internal medicine. He interconnects Serine racemase, Neurotransmission, Ephrin, Cell biology and Neuroplasticity in the investigation of issues within Synaptic plasticity. His Epilepsy research includes themes of Inflammation and Hypothermia, Anesthesia.

Between 2006 and 2021, his most popular works were:

  • EphrinBs Regulate d-Serine Synthesis and Release in Astrocytes (64 citations)
  • Post-traumatic seizure susceptibility is attenuated by hypothermia therapy. (61 citations)
  • Phosphodiesterase Inhibition Rescues Chronic Cognitive Deficits Induced by Traumatic Brain Injury (58 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Enzyme
  • Biochemistry

Thomas J. Sick focuses on Hippocampus, Neuroscience, Sham surgery, Post-traumatic seizure and Hypothermia. His Hippocampus study introduces a deeper knowledge of Internal medicine. His Neuroscience research integrates issues from Synaptic plasticity, Serine racemase, Postsynaptic potential and Neurotransmission.

His study in Sham surgery intersects with areas of studies such as Inflammation, Anesthesia, Dentate gyrus and Epilepsy.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Increased seizure duration and slowed potassium kinetics in mice lacking aquaporin-4 water channels.

Devin K. Binder;Xiaoming Yao;Zsolt Zador;Thomas J. Sick.
Glia (2006)

349 Citations

Rapid preconditioning protects rats against ischemic neuronal damage after 3 but not 7 days of reperfusion following global cerebral ischemia

Miguel A. Pérez-Pinzón;Guang Ping Xu;W. Dalton Dietrich;Myron Rosenthal.
Journal of Cerebral Blood Flow and Metabolism (1997)

306 Citations

εPKC Is Required for the Induction of Tolerance by Ischemic and NMDA-Mediated Preconditioning in the Organotypic Hippocampal Slice

Ami P. Raval;Kunjan R. Dave;Daria Mochly-Rosen;Thomas J. Sick.
The Journal of Neuroscience (2003)

254 Citations

Surviving Hypoxia: Mechanisms of Control and Adaptation

Thomas J. Sick;Peter W. Hochachka.
(1993)

241 Citations

Anoxic preconditioning in hippocampal slices: role of adenosine

Miguel Perez-Pinzon;P. L. Mumford;M. Rosenthal;Thomas Sick.
Neuroscience (1996)

237 Citations

Protein aggregation after focal brain ischemia and reperfusion.

Bing Ren Hu;Shorena Janelidze;Myron D. Ginsberg;Raul Busto.
Journal of Cerebral Blood Flow and Metabolism (2001)

209 Citations

Downregulation of sodium channels during anoxia: a putative survival strategy of turtle brain.

M. A. Perez-Pinzon;M. Rosenthal;T. J. Sick;P. L. Lutz.
American Journal of Physiology-regulatory Integrative and Comparative Physiology (1992)

184 Citations

Cytochrome C Is Released from Mitochondria Into the Cytosol after Cerebral Anoxia or Ischemia

Miguel A. Pérez-Pinzón;Guang Ping Xu;James Born;José Lorenzo.
Journal of Cerebral Blood Flow and Metabolism (1999)

179 Citations

Brain potassium ion homeostasis, anoxia, and metabolic inhibition in turtles and rats

T. J. Sick;M. Rosenthal;J. C. LaManna;P. L. Lutz.
American Journal of Physiology-regulatory Integrative and Comparative Physiology (1982)

166 Citations

Chronic failure in the maintenance of long-term potentiation following fluid percussion injury in the rat

Matthew J Sanders;Thomas J Sick;Miguel A Perez-Pinzon;W.Dalton Dietrich.
Brain Research (2000)

153 Citations

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