Her scientific interests lie mostly in Neuroscience, Amygdala, Neurotransmission, Basolateral amygdala and Endocrinology. Patricia Shinnick-Gallagher interconnects Biophysics and Metabotropic glutamate receptor in the investigation of issues within Neuroscience. The study incorporates disciplines such as Long-term potentiation and Kindling in addition to Amygdala.
Her research investigates the link between Neurotransmission and topics such as Postsynaptic potential that cross with problems in Electrophysiology. Her Basolateral amygdala research is multidisciplinary, incorporating elements of Glutamate receptor and Intracellular. Patricia Shinnick-Gallagher has researched Endocrinology in several fields, including Internal medicine, T-type calcium channel, Calcium channel and Cell biology.
Patricia Shinnick-Gallagher mainly investigates Neuroscience, Amygdala, Internal medicine, Endocrinology and Excitatory postsynaptic potential. Her Neuroscience research includes elements of Postsynaptic potential, Neurotransmission and Metabotropic glutamate receptor. Her research integrates issues of NMDA receptor, Long-term potentiation, Nucleus and Kindling in her study of Amygdala.
She works mostly in the field of Internal medicine, limiting it down to topics relating to Membrane potential and, in certain cases, Neuromuscular junction and Tetrodotoxin. The Propranolol research she does as part of her general Endocrinology study is frequently linked to other disciplines of science, such as Soleus muscle, therefore creating a link between diverse domains of science. Her studies deal with areas such as AMPA receptor, Glutamatergic and Medial vestibular nucleus as well as Excitatory postsynaptic potential.
Neuroscience, Amygdala, Long-term potentiation, Synaptic plasticity and Neurotransmission are her primary areas of study. Her study in Neuroscience is interdisciplinary in nature, drawing from both Glutamate receptor and Receptor. Patricia Shinnick-Gallagher usually deals with Glutamate receptor and limits it to topics linked to Dopamine and Inhibitory postsynaptic potential, Anesthesia, Metabotropic glutamate receptor 1 and Metabotropic receptor.
Her work deals with themes such as Metabotropic glutamate receptor and Kindling, which intersect with Amygdala. Neurotransmission is frequently linked to Excitatory postsynaptic potential in her study. The various areas that Patricia Shinnick-Gallagher examines in her Excitatory postsynaptic potential study include Glutamatergic and Postsynaptic potential.
Patricia Shinnick-Gallagher spends much of her time researching Amygdala, Neuroscience, Long-term potentiation, Neurotransmission and Internal medicine. Her Neuroscience research is multidisciplinary, relying on both Receptor and Corticotropin-releasing hormone receptor 1. In her work, Corticotropin-releasing hormone, Synapse and Hippocampus is strongly intertwined with Synaptic plasticity, which is a subfield of Long-term potentiation.
Her Neurotransmission study incorporates themes from Fear conditioning and Excitatory postsynaptic potential. Her Internal medicine study frequently links to adjacent areas such as Endocrinology. Her Basolateral amygdala study combines topics in areas such as Calcium, T-type calcium channel, P-type calcium channel, Cell biology and Glucocorticoid receptor.
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Fear conditioning induces a lasting potentiation of synaptic currents in vitro
M. G. McKernan;P. Shinnick-Gallagher.
Inhibitory transmission in the basolateral amygdala.
Donald G. Rainnie;Eftihia K. Asprodini;Patricia Shinnick-Gallagher.
Journal of Neurophysiology (1991)
Intracellular recordings from morphologically identified neurons of the basolateral amygdala
D. G. Rainnie;E. K. Asprodini;P. Shinnick-Gallagher.
Journal of Neurophysiology (1993)
Synaptic physiology of central CRH system.
Joel P. Gallagher;Luis F. Orozco-Cabal;Jie Liu;Patricia Shinnick-Gallagher.
European Journal of Pharmacology (2008)
Corticotropin-Releasing Factor and Urocortin I Modulate Excitatory Glutamatergic Synaptic Transmission
Jie Liu;Baojian Yu;Volker Neugebauer;Dimitri E. Grigoriadis.
The Journal of Neuroscience (2004)
Glucocorticoids alter calcium conductances and calcium channel subunit expression in basolateral amygdala neurons.
Henk Karst;Suresh Nair;Els Velzing;Lisette Rumpff-van Essen.
European Journal of Neuroscience (2002)
Differential actions of corticotropin releasing factor on basolateral and central amygdaloid neurones, in vitro.
D. G. Rainnie;B. J.H. Fernhout;P. Shinnick-Gallagher.
Journal of Pharmacology and Experimental Therapeutics (1992)
Epileptogenesis In Vivo Enhances the Sensitivity of Inhibitory Presynaptic Metabotropic Glutamate Receptors in Basolateral Amygdala Neurons In Vitro
V Neugebauer;N B Keele;P Shinnick-Gallagher.
The Journal of Neuroscience (1997)
Spontaneous epileptiform activity and alteration of GABA- and of NMDA-mediated neurotransmission in amygdala neurons kindled in vivo.
Po-Wu Gean;Patricia Shinnick-Gallagher;Anthony C. Anderson.
Brain Research (1989)
Kindling-induced long-lasting changes in synaptic transmission in the basolateral amygdala.
D. G. Rainnie;E. K. Asprodini;P. Shinnick-Gallagher.
Journal of Neurophysiology (1992)
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