Her primary areas of investigation include Biochemistry, Transferrin, Cell biology, Hepcidin and Transferrin receptor. Her studies examine the connections between Biochemistry and genetics, as well as such issues in Mononuclear phagocyte system, with regards to Metabolism, Phagocytosis and Erythrophagocytosis. Her Transferrin research integrates issues from Hemochromatosis, Extracellular and Ferritin.
Particularly relevant to Endosome is her body of work in Cell biology. Her work investigates the relationship between Hepcidin and topics such as Homeostasis that intersect with problems in Anemia of chronic disease and Anemia. Her Transferrin receptor research includes themes of Saccharomyces cerevisiae Proteins and Iron deficiency.
Marianne Wessling-Resnick focuses on Biochemistry, Cell biology, Transferrin, Endocrinology and Internal medicine. Her Biochemistry study incorporates themes from Hemochromatosis and Hepcidin. Her study looks at the relationship between Cell biology and fields such as Endocytic cycle, as well as how they intersect with chemical problems.
Her Transferrin research is mostly focused on the topic Transferrin receptor. Her research in Endocrinology intersects with topics in Hereditary hemochromatosis, Manganese and Iron deficiency. Marianne Wessling-Resnick has researched Internal medicine in several fields, including Genetic model and In vivo.
Marianne Wessling-Resnick mainly focuses on Hepcidin, Cell biology, Endocrinology, Internal medicine and DMT1. Marianne Wessling-Resnick interconnects Genistein, Transferrin saturation, Small molecule, Micronutrient and STAT3 in the investigation of issues within Hepcidin. Her research integrates issues of Caspase 12, Microglia, Neuroinflammation, Transferrin receptor and In vivo in her study of Cell biology.
Her biological study spans a wide range of topics, including Hereditary hemochromatosis, Genetic model and Anemia. Her DMT1 study combines topics in areas such as Iron transport, Transferrin and Iron uptake. Her Biochemistry study frequently draws connections to other fields, such as Hemochromatosis.
The scientist’s investigation covers issues in Iron deficiency, Cell biology, DMT1, Transporter and Neuroinflammation. Her work carried out in the field of Iron deficiency brings together such families of science as Caspase 3, Oxidative stress, Glucose-regulated protein, Caspase 12 and Neurotransmitter. Her studies deal with areas such as Transferrin receptor and Apoptosis, Programmed cell death as well as Cell biology.
Her DMT1 research is classified as research in Biochemistry. While working in this field, Marianne Wessling-Resnick studies both Biochemistry and Hinokitiol. Her study explores the link between Neuroinflammation and topics such as Microglia that cross with problems in Secretion, Neurodegeneration, Transferrin and Ferritin.
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Chemistry and biology of eukaryotic iron metabolism.
Philip Aisen;Caroline Enns;Marianne Wessling-Resnick.
The International Journal of Biochemistry & Cell Biology (2001)
Iron release from macrophages after erythrophagocytosis is up-regulated by ferroportin 1 overexpression and down-regulated by hepcidin
Mitchell D. Knutson;Mohamed Oukka;Lindsey M. Koss;Fikret Aydemir.
Proceedings of the National Academy of Sciences of the United States of America (2005)
Iron Homeostasis and the Inflammatory Response
Annual Review of Nutrition (2010)
Iron Metabolism in the Reticuloendothelial System
Mitchell Knutson;Marianne Wessling-Resnick.
Critical Reviews in Biochemistry and Molecular Biology (2003)
Iron metabolism and the innate immune response to infection.
Erin E. Johnson;Marianne Wessling-Resnick.
Microbes and Infection (2012)
Regulation of transferrin receptor 2 protein levels by transferrin.
Aeisha Robb;Marianne Wessling-Resnick.
Iron loading and erythrophagocytosis increase ferroportin 1 (FPN1) expression in J774 macrophages.
Mitchell D. Knutson;Mohammad R. Vafa;David J. Haile;Marianne Wessling-Resnick.
Hepcidin Regulation of Iron Transport
James F. Collins;Marianne Wessling-Resnick;Mitchell D. Knutson.
Journal of Nutrition (2008)
Wortmannin alters the transferrin receptor endocytic pathway in vivo and in vitro.
D J Spiro;W Boll;T Kirchhausen;M Wessling-Resnick.
Molecular Biology of the Cell (1996)
Attenuated inflammatory responses in hemochromatosis reveal a role for iron in the regulation of macrophage cytokine translation.
Lijian Wang;Erin E. Johnson;Hai Ning Shi;W. Allan Walker.
Journal of Immunology (2008)
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