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Neuroscience

D-Index
74
Citations
14330
World Ranking
2154
National Ranking
1022

Overview

Alvin J. Beitz is affiliated with the University of Minnesota in the United States. Their research primarily focuses on medicine and neuroscience, with particular attention to subfields such as physiology, cellular and molecular neuroscience, molecular biology, and pharmacology.

Their recent published work includes two papers addressing aspects of pain mechanisms and pharmacological regulation in neuropathic pain models. The papers are:

  • Intrathecal interleukin-1β decreases sigma-1 receptor expression in spinal astrocytes in a murine model of neuropathic pain, 2021, Biomedicine & Pharmacotherapy
  • Phase-specific differential regulation of mechanical allodynia in a murine model of neuropathic pain by progesterone, 2023, Frontiers in Pharmacology

These publications highlight topics related to pain mechanisms and treatments, neuropeptides and animal physiology, pharmacological receptor mechanisms and effects, and the pharmacological effects of natural compounds.

Frequent coauthors collaborating with Beitz include:

  • Sheu-Ran Choi
  • Jang-Hern Lee
  • Ho Jae Han
  • Dae-Hyun Roh
  • Ji-Young Moon

Beitz's work is published in venues such as Biomedicine & Pharmacotherapy and Frontiers in Pharmacology, reflecting their engagement with journals in biomedical and pharmacological research.

The main research topics associated with Alvin J. Beitz's work are:

  • Pain Mechanisms and Treatments
  • Neuropeptides and Animal Physiology
  • Pharmacological Receptor Mechanisms and Effects
  • Pharmacological Effects of Natural Compounds

Overall, the research profile of Alvin J. Beitz centers on understanding the molecular and cellular basis of neuropathic pain and examining the pharmacological modulation of this condition through experimental models.

Best Publications

  • The organization of afferent projections to the midbrain periaqueductal gray of the rat

    A.J. Beitz

  • Extracellular amino acid concentrations in the dorsal spinal cord of freely moving rats following veratridine and nociceptive stimulation

    Stephen R. Skilling;David H. Smullin;Alvin J. Beitz;Alice A. Larson

  • Bee venom injection into an acupuncture point reduces arthritis associated edema and nociceptive responses.

    Young Bae Kwon;Jae Dong Lee;Hye Jung Lee;Ho Jae Han

  • Functional Interactions between Tumor and Peripheral Nerve: Morphology, Algogen Identification, and Behavioral Characterization of a New Murine Model of Cancer Pain

    Paul W. Wacnik;Laura J. Eikmeier;Timothy R. Ruggles;Margaret L. Ramnaraine

  • The distribution of brain-stem and spinal cord nuclei associated with different frequencies of electroacupuncture analgesia

    Lee Jang-Hern;Alvin J. Beitz

  • The water-soluble fraction of bee venom produces antinociceptive and anti-inflammatory effects on rheumatoid arthritis in rats.

    Young Bae Kwon;Hye Jung Lee;Ho Jae Han;Woung Chon Mar

  • Nitric oxide synthase-containing neural processes on large cerebral arteries and cerebral microvessels.

    Costantino Iadecola;Alvin J. Beitz;Waleed Renno;Xiaohong Xu

  • The nuclei of origin of brain stem enkephalin and substance P projections to the rodent nucleus raphe magnus.

    A.J. Beitz

  • Intrathecal injection of the ς1 receptor antagonist BD1047 blocks both mechanical allodynia and increases in spinal NR1 expression during the induction phase of rodent neuropathic pain

    Dae Hyun Roh;Hyun Woo Kim;Seo Yeon Yoon;Hyoung Sig Seo

  • The sites of origin brain stem neurotensin and serotonin projections to the rodent nucleus raphe magnus

    Unknown

  • The midbrain periaqueductal gray in the rat. I. Nuclear volume, cell number, density, orientation, and regional subdivisions.

    Alvin J. Beitz

  • Nitric oxide synthase immunoreactive neurons anatomically define a longitudinal dorsolateral column within the midbrain periaqueductal gray of the rat: analysis using laser confocal microscopy

    Donna Onstott;Bernd Mayer;Alvin J. Beitz

  • Activation of the spinal sigma‐1 receptor enhances NMDA‐induced pain via PKC‐ and PKA‐dependent phosphorylation of the NR1 subunit in mice

    Kim Hw;Roh Dh;Yoon Sy;Seo Hs

  • The periaqueductal gray-raphe magnus projection contains somatostatin, neurotensin and serotonin but not cholecystokinin.

    Alvin J. Beitz;R. David Shepard;William E. Wells

  • The periaqueductal gray projections to the rat spinal trigeminal, raphe magnus, gigantocellular pars alpha and paragigantocellular nuclei arise from separate neurons

    Alvin J. Beitz;Mary A. Mullett;Lorraine L. Weiner

  • Monoclonal antibody specific for carbodiimide-fixed glutamate: immunocytochemical localization in the rat CNS.

    James E. Madl;Alice A. Larson;Alvin J. Beitz

  • Excitatory amino acid binding sites in the trigeminal principal sensory and spinal trigeminal nuclei of the rat.

    Sara J. Tallaksen-Greene;Anne B. Young;John B. Penney;Alvin J. Beitz

  • Acupoint Stimulation Using Bee Venom Attenuates Formalin-Induced Pain Behavior and Spinal Cord Fos Expression in Rats

    Hyun Woo Kim;Young Bae Kwon;Tae Won Ham;Dae Hyun Roh

  • Glycine potentiates strychnine-induced convulsions: role of NMDA receptors

    AA Larson;AJ Beitz

  • Animal models of cancer pain.

    Cholawat Pacharinsak;Alvin Beitz

  • Intrathecal treatment with σ1 receptor antagonists reduces formalin-induced phosphorylation of NMDA receptor subunit 1 and the second phase of formalin test in mice

    Hyun Woo Kim;Young Bae Kwon;Dae Hyun Roh;Seo Yeon Yoon

Frequent Co-Authors

Seo Yeon Yoon
Seo Yeon Yoon Gladstone Institutes
Alice A. Larson
Alice A. Larson University of Minnesota
Donald A. Simone
Donald A. Simone University of Minnesota
George L. Wilcox
George L. Wilcox University of Minnesota
Seog Bae Oh
Seog Bae Oh Seoul National University
Michael P. Murtaugh
Michael P. Murtaugh University of Minnesota
Costantino Iadecola
Costantino Iadecola Cornell University
Jin Mo Chung
Jin Mo Chung The University of Texas Medical Branch at Galveston
Patrick W. Mantyh
Patrick W. Mantyh University of Arizona
William H. Frey
William H. Frey HealthPartners

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