D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 53 Citations 8,715 138 World Ranking 8389 National Ranking 3767

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Cancer

Pathology, Lung, Idiopathic pulmonary fibrosis, Immunology and Epithelial–mesenchymal transition are his primary areas of study. His Pathology research includes themes of Multiple sclerosis, Tight junction, Lung disease and Adult stem cell. His Lung research incorporates elements of Histopathology, Glutathione and Lung morphogenesis.

His Idiopathic pulmonary fibrosis research is multidisciplinary, relying on both Fibrosis, Pulmonary fibrosis, Median survival and Interstitial lung disease. The concepts of his Immunology study are interwoven with issues in Pulmonary alveolus, Disease and Respiratory system. His work carried out in the field of Epithelial–mesenchymal transition brings together such families of science as Myofibroblast and Cell biology.

His most cited work include:

  • Induction of Epithelial-Mesenchymal Transition in Alveolar Epithelial Cells by Transforming Growth Factor-β1 : Potential Role in Idiopathic Pulmonary Fibrosis (778 citations)
  • TGF-β-induced EMT: mechanisms and implications for fibrotic lung disease (759 citations)
  • Inhibition of Wnt/β-catenin/CREB binding protein (CBP) signaling reverses pulmonary fibrosis (360 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Cell biology, Lung, Cancer research, Molecular biology and Pathology. Zea Borok has researched Cell biology in several fields, including Epithelial–mesenchymal transition, Epithelium, Alveolar Epithelium, Cellular differentiation and Alveolar epithelial cell. His research in Cellular differentiation tackles topics such as Stem cell which are related to areas like Immunology.

His Lung study combines topics in areas such as Fibrosis, Glutathione and Respiratory system. As a member of one scientific family, Zea Borok mostly works in the field of Molecular biology, focusing on Gene expression and, on occasion, Transdifferentiation. The various areas that Zea Borok examines in his Pathology study include Lung injury and Idiopathic pulmonary fibrosis.

He most often published in these fields:

  • Cell biology (44.84%)
  • Lung (18.25%)
  • Cancer research (15.48%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (44.84%)
  • Cancer research (15.48%)
  • Myofibroblast (5.56%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Cell biology, Cancer research, Myofibroblast, Lung and Claudin. Zea Borok interconnects Cell, Transcriptome, Transcription factor and Cellular differentiation in the investigation of issues within Cell biology. His research in Cancer research intersects with topics in Carcinogenesis, Cancer, Adenocarcinoma, Idiopathic pulmonary fibrosis and Lung cancer.

His Idiopathic pulmonary fibrosis study also includes fields such as

  • Fibroblast which is related to area like Cell type,
  • Receptor which connect with WNT5A. His Myofibroblast study deals with Mesenchymal stem cell intersecting with Fibrosis. His Lung research is multidisciplinary, incorporating elements of Epithelium, Wnt signaling pathway, Molecular Profile, Human Induced Pluripotent Stem Cells and Cohort.

Between 2018 and 2021, his most popular works were:

  • Circulating angiotensin peptides levels in Acute Respiratory Distress Syndrome correlate with clinical outcomes: A pilot study. (45 citations)
  • LINC00261 Is an Epigenetically Regulated Tumor Suppressor Essential for Activation of the DNA Damage Response (40 citations)
  • Epithelial Vegfa Specifies a Distinct Endothelial Population in the Mouse Lung (36 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Cancer

Zea Borok mainly focuses on Cell biology, Cancer research, Myofibroblast, Transcriptome and Gene. His Cell biology research incorporates themes from Alveolar epithelial cell, Pulmonary fibrosis and Cellular differentiation. His biological study spans a wide range of topics, including Carcinogenesis, Lung cancer and DNA methylation.

The Myofibroblast study combines topics in areas such as Mesenchymal stem cell, Pathogenesis and Lung, Idiopathic pulmonary fibrosis. Within one scientific family, Zea Borok focuses on topics pertaining to Fibrosis under Mesenchymal stem cell, and may sometimes address concerns connected to Cell type. The concepts of his Lung study are interwoven with issues in Endothelial stem cell, Epithelium, Angiogenesis and Basement membrane.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

TGF-β-induced EMT: mechanisms and implications for fibrotic lung disease

Brigham C. Willis;Zea Borok.
American Journal of Physiology-lung Cellular and Molecular Physiology (2007)

1218 Citations

Induction of Epithelial-Mesenchymal Transition in Alveolar Epithelial Cells by Transforming Growth Factor-β1 : Potential Role in Idiopathic Pulmonary Fibrosis

Brigham C. Willis;Brigham C. Willis;Brigham C. Willis;Janice M. Liebler;Katherine Luby-Phelps;Andrew G. Nicholson.
American Journal of Pathology (2005)

1045 Citations

Epithelial origin of myofibroblasts during fibrosis in the lung

Brigham C. Willis;Roland M. duBois;Zea Borok.
Proceedings of the American Thoracic Society (2006)

541 Citations

Systemic deficiency of glutathione in cystic fibrosis.

J. H. Roum;R. Buhl;N. G. McElvaney;Z. Borok.
Journal of Applied Physiology (1993)

432 Citations

Inhibition of Wnt/β-catenin/CREB binding protein (CBP) signaling reverses pulmonary fibrosis

William R. Henderson;Emil Y. Chi;Xin Ye;Cu Nguyen.
Proceedings of the National Academy of Sciences of the United States of America (2010)

417 Citations

Wnt5a Participates in Distal Lung Morphogenesis

Changgong Li;Jing Xiao;Khadija Hormi;Zea Borok.
Developmental Biology (2002)

392 Citations

Pulmonary Langerhans cell granulomatosis (histiocytosis X). A clinicopathologic study of 48 cases.

William D. Travis;Zea Borok;James H. Roum;Jun Zhang.
The American Journal of Surgical Pathology (1993)

380 Citations

Keratinocyte growth factor modulates alveolar epithelial cell phenotype in vitro: Expression of aquaporin 5

Zea Borok;Richard L. Lubman;Spencer I. Danto;Xiao Ling Zhang.
American Journal of Respiratory Cell and Molecular Biology (1998)

271 Citations

Interactions between β-catenin and transforming growth factor-β signaling pathways mediate epithelial-mesenchymal transition and are dependent on the transcriptional co-activator cAMP-response element-binding protein (CREB)-binding protein (CBP).

Beiyun Zhou;Yixin Liu;Michael Kahn;David K. Ann.
Journal of Biological Chemistry (2012)

270 Citations

Effect of glutathione aerosol on oxidant-antioxidant imbalance in idiopathic pulmonary fibrosis.

Z Borok;R Buhl;G J Grimes;A D Bokser.
The Lancet (1991)

269 Citations

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