Yasuhide Hayashi mainly investigates Molecular biology, Cancer research, Genetics, Myeloid leukemia and Gene. His studies in Molecular biology integrate themes in fields like Fusion gene, Fusion transcript, Chromosomal translocation, Mutation and Chromosome 16. His Cancer research research incorporates themes from Carcinogenesis, Tumor suppressor gene, Leukemia, Immunology and Loss of heterozygosity.
When carried out as part of a general Genetics research project, his work on Germline mutation and Exome sequencing is frequently linked to work in Gene nomenclature, Septin and Septin complex, therefore connecting diverse disciplines of study. His studies deal with areas such as Gene rearrangement, Gene product, RUNX1T1 and Transcription factor as well as Myeloid leukemia. His Nonsense mutation, Fungal protein and Gene mapping study, which is part of a larger body of work in Gene, is frequently linked to SEPT2, bridging the gap between disciplines.
Yasuhide Hayashi mainly focuses on Cancer research, Internal medicine, Leukemia, Molecular biology and Gene. His Cancer research research is multidisciplinary, incorporating perspectives in Mutation, Carcinogenesis, Immunology, Lymphoma and Neuroblastoma. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology, Oncology and NPM1.
His Leukemia study incorporates themes from Myeloid, Gene rearrangement and Myeloid leukemia. His Molecular biology research is multidisciplinary, relying on both Cell culture, Nonsense mutation, Missense mutation, Chromosomal translocation and Fusion gene. His Gene study is concerned with the larger field of Genetics.
Yasuhide Hayashi spends much of his time researching Cancer research, Internal medicine, Oncology, Leukemia and Gene. The concepts of his Cancer research study are interwoven with issues in Gene expression profiling, Mutation, Transcriptome, Childhood leukemia and Chromosome abnormality. His work on Survival rate and Myeloid leukemia is typically connected to In patient as part of general Internal medicine study, connecting several disciplines of science.
His research investigates the connection between Oncology and topics such as Transplantation that intersect with problems in Stem cell and Fusion gene. His work deals with themes such as Hematopoietic stem cell transplantation, Myeloid, Hematology, Lymphoma and Down syndrome, which intersect with Leukemia. Gene is a subfield of Genetics that Yasuhide Hayashi tackles.
His primary areas of investigation include Internal medicine, Cancer research, Oncology, Myeloid leukemia and Leukemia. His Internal medicine research incorporates elements of Gastroenterology, Immunology and NPM1. The study incorporates disciplines such as Exome sequencing, Fusion gene, Frameshift mutation and Lymphoma in addition to Cancer research.
His work carried out in the field of Myeloid leukemia brings together such families of science as Survival rate, Down syndrome and GATA1. His biological study spans a wide range of topics, including Myeloid and Gene rearrangement. His Gene study frequently involves adjacent topics like Molecular biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Oncogenic mutations of ALK kinase in neuroblastoma.
Yuyan Chen;Junko Takita;Young Lim Choi;Motohiro Kato.
Nature (2008)
Charcot-Marie-Tooth Disease Type 2A Caused by Mutation in a Microtubule Motor KIF1Bβ
Chunjie Zhao;Junko Takita;Yosuke Tanaka;Mitsutoshi Setou.
Cell (2001)
Frequent inactivation of A20 in B-cell lymphomas
Motohiro Kato;Masashi Sanada;Itaru Kato;Yasuharu Sato.
Nature (2009)
Gain-of-function of mutated C-CBL tumour suppressor in myeloid neoplasms.
Masashi Sanada;Takahiro Suzuki;Lee-Yung Shih;Makoto Otsu.
Nature (2009)
LCX, Leukemia-associated Protein with a CXXC Domain, Is Fused to MLL in Acute Myeloid Leukemia with Trilineage Dysplasia Having t(10;11)(q22;q23)
Ryoichi Ono;Tomohiko Taki;Takeshi Taketani;Masafumi Taniwaki.
Cancer Research (2002)
An RNA-binding Protein Gene, TLS/FUS , Is Fused to ERG in Human Myeloid Leukemia with t(16;21) Chromosomal Translocation
Hitoshi Ichikawa;Kimiko Shimizu;Yasuhide Hayashi;Misao Ohki.
Cancer Research (1994)
The landscape of somatic mutations in Down syndrome-related myeloid disorders
Kenichi Yoshida;Tsutomu Toki;Yusuke Okuno;Rika Kanezaki.
Nature Genetics (2013)
Adenoviral E1A-associated protein p300 is involved in acute myeloid leukemia with t(11;22)(q23;q13).
Kohmei Ida;Issay Kitabayashi;Tomohiko Taki;Masafumi Taniwaki.
Blood (1997)
The t(11;16)(q23;p13) translocation in myelodysplastic syndrome fuses the MLL gene to the CBP gene.
Tomohiko Taki;Masahiro Sako;Masahiro Tsuchida;Yasuhide Hayashi.
Blood (1997)
FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy
Takeshi Taketani;Tomohiko Taki;Kanji Sugita;Yoshiyuki Furuichi.
Blood (2003)
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Publications: 47
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