D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 74 Citations 18,823 438 World Ranking 15193 National Ranking 503
Biology and Biochemistry D-index 74 Citations 18,662 429 World Ranking 3517 National Ranking 239

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Mutation

Yasuhide Hayashi mainly investigates Molecular biology, Cancer research, Genetics, Myeloid leukemia and Gene. His studies in Molecular biology integrate themes in fields like Fusion gene, Fusion transcript, Chromosomal translocation, Mutation and Chromosome 16. His Cancer research research incorporates themes from Carcinogenesis, Tumor suppressor gene, Leukemia, Immunology and Loss of heterozygosity.

When carried out as part of a general Genetics research project, his work on Germline mutation and Exome sequencing is frequently linked to work in Gene nomenclature, Septin and Septin complex, therefore connecting diverse disciplines of study. His studies deal with areas such as Gene rearrangement, Gene product, RUNX1T1 and Transcription factor as well as Myeloid leukemia. His Nonsense mutation, Fungal protein and Gene mapping study, which is part of a larger body of work in Gene, is frequently linked to SEPT2, bridging the gap between disciplines.

His most cited work include:

  • Oncogenic mutations of ALK kinase in neuroblastoma. (686 citations)
  • Charcot-Marie-Tooth Disease Type 2A Caused by Mutation in a Microtubule Motor KIF1Bβ (651 citations)
  • Frequent inactivation of A20 in B-cell lymphomas (453 citations)

What are the main themes of his work throughout his whole career to date?

Yasuhide Hayashi mainly focuses on Cancer research, Internal medicine, Leukemia, Molecular biology and Gene. His Cancer research research is multidisciplinary, incorporating perspectives in Mutation, Carcinogenesis, Immunology, Lymphoma and Neuroblastoma. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology, Oncology and NPM1.

His Leukemia study incorporates themes from Myeloid, Gene rearrangement and Myeloid leukemia. His Molecular biology research is multidisciplinary, relying on both Cell culture, Nonsense mutation, Missense mutation, Chromosomal translocation and Fusion gene. His Gene study is concerned with the larger field of Genetics.

He most often published in these fields:

  • Cancer research (36.18%)
  • Internal medicine (27.65%)
  • Leukemia (24.19%)

What were the highlights of his more recent work (between 2014-2021)?

  • Cancer research (36.18%)
  • Internal medicine (27.65%)
  • Oncology (14.06%)

In recent papers he was focusing on the following fields of study:

Yasuhide Hayashi spends much of his time researching Cancer research, Internal medicine, Oncology, Leukemia and Gene. The concepts of his Cancer research study are interwoven with issues in Gene expression profiling, Mutation, Transcriptome, Childhood leukemia and Chromosome abnormality. His work on Survival rate and Myeloid leukemia is typically connected to In patient as part of general Internal medicine study, connecting several disciplines of science.

His research investigates the connection between Oncology and topics such as Transplantation that intersect with problems in Stem cell and Fusion gene. His work deals with themes such as Hematopoietic stem cell transplantation, Myeloid, Hematology, Lymphoma and Down syndrome, which intersect with Leukemia. Gene is a subfield of Genetics that Yasuhide Hayashi tackles.

Between 2014 and 2021, his most popular works were:

  • Integrated genetic and epigenetic analysis defines novel molecular subgroups in rhabdomyosarcoma (81 citations)
  • Haploinsufficiency of TNFAIP3 (A20) by germline mutation is involved in autoimmune lymphoproliferative syndrome (62 citations)
  • Recurrent SPI1 (PU.1) fusions in high-risk pediatric T cell acute lymphoblastic leukemia (53 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Mutation

His primary areas of investigation include Internal medicine, Cancer research, Oncology, Myeloid leukemia and Leukemia. His Internal medicine research incorporates elements of Gastroenterology, Immunology and NPM1. The study incorporates disciplines such as Exome sequencing, Fusion gene, Frameshift mutation and Lymphoma in addition to Cancer research.

His work carried out in the field of Myeloid leukemia brings together such families of science as Survival rate, Down syndrome and GATA1. His biological study spans a wide range of topics, including Myeloid and Gene rearrangement. His Gene study frequently involves adjacent topics like Molecular biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Oncogenic mutations of ALK kinase in neuroblastoma.

Yuyan Chen;Junko Takita;Young Lim Choi;Motohiro Kato.
Nature (2008)

978 Citations

Charcot-Marie-Tooth Disease Type 2A Caused by Mutation in a Microtubule Motor KIF1Bβ

Chunjie Zhao;Junko Takita;Yosuke Tanaka;Mitsutoshi Setou.
Cell (2001)

883 Citations

Frequent inactivation of A20 in B-cell lymphomas

Motohiro Kato;Masashi Sanada;Itaru Kato;Yasuharu Sato.
Nature (2009)

634 Citations

Gain-of-function of mutated C-CBL tumour suppressor in myeloid neoplasms.

Masashi Sanada;Takahiro Suzuki;Lee-Yung Shih;Makoto Otsu.
Nature (2009)

455 Citations

LCX, Leukemia-associated Protein with a CXXC Domain, Is Fused to MLL in Acute Myeloid Leukemia with Trilineage Dysplasia Having t(10;11)(q22;q23)

Ryoichi Ono;Tomohiko Taki;Takeshi Taketani;Masafumi Taniwaki.
Cancer Research (2002)

379 Citations

An RNA-binding Protein Gene, TLS/FUS , Is Fused to ERG in Human Myeloid Leukemia with t(16;21) Chromosomal Translocation

Hitoshi Ichikawa;Kimiko Shimizu;Yasuhide Hayashi;Misao Ohki.
Cancer Research (1994)

350 Citations

The landscape of somatic mutations in Down syndrome-related myeloid disorders

Kenichi Yoshida;Tsutomu Toki;Yusuke Okuno;Rika Kanezaki.
Nature Genetics (2013)

346 Citations

Adenoviral E1A-associated protein p300 is involved in acute myeloid leukemia with t(11;22)(q23;q13).

Kohmei Ida;Issay Kitabayashi;Tomohiko Taki;Masafumi Taniwaki.
Blood (1997)

345 Citations

The t(11;16)(q23;p13) translocation in myelodysplastic syndrome fuses the MLL gene to the CBP gene.

Tomohiko Taki;Masahiro Sako;Masahiro Tsuchida;Yasuhide Hayashi.
Blood (1997)

342 Citations

FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy

Takeshi Taketani;Tomohiko Taki;Kanji Sugita;Yoshiyuki Furuichi.
Blood (2003)

292 Citations

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