D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 47 Citations 5,934 160 World Ranking 3748 National Ranking 1700

Overview

What is he best known for?

The fields of study he is best known for:

  • Retina
  • Gene
  • Internal medicine

Samuel M. Wu mostly deals with Retina, Neuroscience, Biophysics, Anatomy and Inner plexiform layer. The various areas that he examines in his Retina study include Endocrinology, Postsynaptic potential, Retinal, Neurotransmission and Synapse. He specializes in Biophysics, namely Depolarization.

His Anatomy research includes themes of Glaucoma and Outer nuclear layer. Samuel M. Wu has researched Inner plexiform layer in several fields, including Axon, Receptive field and Inhibitory postsynaptic potential, Excitatory postsynaptic potential. He works mostly in the field of Excitatory postsynaptic potential, limiting it down to topics relating to Lucifer yellow and, in certain cases, Cell morphology and GABAB receptor.

His most cited work include:

  • Light-Evoked Excitatory and Inhibitory Synaptic Inputs to ON and OFF α Ganglion Cells in the Mouse Retina (189 citations)
  • mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. (185 citations)
  • Functional architecture of synapses in the inner retina: segregation of visual signals by stratification of bipolar cell axon terminals. (154 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Retina, Neuroscience, Biophysics, Retinal and Anatomy. His work carried out in the field of Retina brings together such families of science as Receptive field, Neurotransmission and Cell biology. His biological study spans a wide range of topics, including Light intensity, Lucifer yellow and Glutamate receptor.

His Glutamate receptor study incorporates themes from Endocrinology and Postsynaptic potential. His Anatomy research incorporates themes from Retinal Rod Photoreceptor Cells, Gap junction and Outer nuclear layer. His work deals with themes such as Voltage clamp, Outer plexiform layer and Axon, which intersect with Inner plexiform layer.

He most often published in these fields:

  • Retina (57.93%)
  • Neuroscience (37.24%)
  • Biophysics (32.41%)

What were the highlights of his more recent work (between 2012-2021)?

  • Retina (57.93%)
  • Retinal ganglion (18.62%)
  • Cell biology (17.93%)

In recent papers he was focusing on the following fields of study:

His main research concerns Retina, Retinal ganglion, Cell biology, Retinal ganglion cell and Ophthalmology. The study incorporates disciplines such as gamma-Aminobutyric acid, Biophysics, Retinal and Anatomy in addition to Retina. His study in Biophysics is interdisciplinary in nature, drawing from both Dendritic spine, Outer plexiform layer, Postsynaptic density, Excitatory postsynaptic potential and Calbindin.

His work on Ganglion is typically connected to Coupling and Sign as part of general Anatomy study, connecting several disciplines of science. His Retinal ganglion study combines topics in areas such as Amacrine cell, Receptive field, TRPV4 and Scotopic vision. The various areas that Samuel M. Wu examines in his Cell biology study include Vertebrate retina, TFEB and Immunology.

Between 2012 and 2021, his most popular works were:

  • mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. (185 citations)
  • Correction: Corrigendum: mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases (96 citations)
  • Elevated intraocular pressure causes inner retinal dysfunction before cell loss in a mouse model of experimental glaucoma. (78 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Retina
  • Internal medicine

Samuel M. Wu spends much of his time researching Cell biology, Retina, Intraocular pressure, Retinal degeneration and Ophthalmology. His Cell biology research incorporates elements of Rhodopsin, Retinal Rod Photoreceptor Cells, Endocrinology and TFEB. His studies in TFEB integrate themes in fields like mTORC1 and Protein kinase B.

His work in Retina covers topics such as Anatomy which are related to areas like Retinal. The concepts of his Intraocular pressure study are interwoven with issues in Electroretinography, Glaucoma and Optic nerve. His Connexin research is multidisciplinary, relying on both gamma-Aminobutyric acid, Inhibitory postsynaptic potential and Ganglion.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases

Michela Palmieri;Rituraj Pal;Hemanth R. Nelvagal;Parisa Lotfi.
Nature Communications (2017)

316 Citations

Light-Evoked Excitatory and Inhibitory Synaptic Inputs to ON and OFF α Ganglion Cells in the Mouse Retina

Ji-Jie Pang;Fan Gao;Samuel M. Wu.
The Journal of Neuroscience (2003)

309 Citations

Correction: Corrigendum: mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases

Michela Palmieri;Rituraj Pal;Hemanth R. Nelvagal;Parisa Lotfi.
Nature Communications (2017)

269 Citations

Functional architecture of synapses in the inner retina: segregation of visual signals by stratification of bipolar cell axon terminals.

Samuel M. Wu;Fan Gao;Bruce R. Maple.
The Journal of Neuroscience (2000)

238 Citations

Input-output relations of the feedback synapse between horizontal cells and cones in the tiger salamander retina.

S. M. Wu.
Journal of Neurophysiology (1991)

148 Citations

BETA2/NeuroD1 Null Mice: A New Model for Transcription Factor-Dependent Photoreceptor Degeneration

Mark E. Pennesi;Jang Hyeon Cho;Zhuo Yang;Schonmei H. Wu.
The Journal of Neuroscience (2003)

135 Citations

Amino acid neurotransmitters in the retina: a functional overview

Samuel M Wu;Bruce R Maple.
Vision Research (1998)

134 Citations

Melatonin enhances horizontal cell sensitivity in salamander retina.

Allan F. Wiechmann;Xiong Li Yang;Samuel M. Wu;Joe G. Hollyfield.
Brain Research (1988)

132 Citations

A quantitative analysis of interactions between photoreceptors in the salamander (Ambystoma) retina.

D Attwell;M Wilson;S M Wu.
The Journal of Physiology (1984)

130 Citations

Feedforward lateral inhibition in retinal bipolar cells: input-output relation of the horizontal cell-depolarizing bipolar cell synapse.

Xiong-Li Yang;Samuel M. Wu.
Proceedings of the National Academy of Sciences of the United States of America (1991)

118 Citations

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