Jaap J. Plomp mainly focuses on Neuromuscular junction, Internal medicine, Endocrinology, Neuroscience and Cell biology. His research integrates issues of Complement membrane attack complex, Acetylcholine and Motor nerve in his study of Neuromuscular junction. His work on Myocyte as part of his general Endocrinology study is frequently connected to Myotonin-protein kinase, thereby bridging the divide between different branches of science.
His research in Neuroscience intersects with topics in Missense mutation, Cortical spreading depression, Migraine and Neurotransmission. His Neurotransmission research incorporates elements of Synapse, Synaptic vesicle and Neurotransmitter. While the research belongs to areas of Cell biology, Jaap J. Plomp spends his time largely on the problem of Complement system, intersecting his research to questions surrounding Ganglioside.
Jaap J. Plomp mostly deals with Neuromuscular junction, Neuroscience, Acetylcholine, Internal medicine and Immunology. Jaap J. Plomp interconnects Postsynaptic potential, Neurotransmitter, Neurotransmission, Neuromuscular transmission and Acetylcholine receptor in the investigation of issues within Neuromuscular junction. His research investigates the connection between Neuromuscular transmission and topics such as Anatomy that intersect with problems in Synaptic vesicle.
His Neuroscience research integrates issues from Familial hemiplegic migraine and Cell biology. His studies deal with areas such as Endocrinology and Cardiology as well as Internal medicine. His Endocrinology study integrates concerns from other disciplines, such as Facial muscles and Receptor tyrosine kinase.
Myasthenia gravis, Acetylcholine receptor, Immunology, Antibody and Neuromuscular junction are his primary areas of study. In his work, Phosphorylation, Molecular biology, Agrin and Affinity maturation is strongly intertwined with Monoclonal antibody, which is a subfield of Myasthenia gravis. Acetylcholine receptor is a subfield of Internal medicine that Jaap J. Plomp studies.
His Neuromuscular junction research incorporates themes from Postsynaptic potential, mdx mouse, Dystrophin, Muscle weakness and Neurotransmission. His Postsynaptic potential research is multidisciplinary, incorporating perspectives in Motor neuron, Skeletal muscle, Neuroscience, Neuromuscular transmission and Cell biology. He combines subjects such as Endocrinology, Facial weakness, Neonatal Fc receptor, Electromyography and In vivo with his study of Muscle weakness.
Jaap J. Plomp mainly investigates Antibody, Antigen, Myasthenia gravis, Sinus rhythm and Inflammation. His Antibody research is multidisciplinary, relying on both Molecular biology, Phosphorylation and Agrin. As part of his studies on Antigen, Jaap J. Plomp often connects relevant subjects like Acetylcholine receptor.
His Sinus rhythm research includes elements of Right atrial and Arrhythmia detection. His Inflammation study combines topics from a wide range of disciplines, such as Autoantibody, Neuromuscular junction, Receptor and Autoimmunity. His Adenoassociated virus research includes a combination of various areas of study, such as Rhythm, Internal medicine and Atrial fibrillation.
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Synaptic Assembly of the Brain in the Absence of Neurotransmitter Secretion
M Verhage;A S Maia;J J Plomp;A B Brussaard.
A Cacna1a Knockin Migraine Mouse Model with Increased Susceptibility to Cortical Spreading Depression
Arn M.J.M van den Maagdenberg;Daniela Pietrobon;Tommaso Pizzorusso;Simon Kaja.
Acquired neuromyotonia: evidence for autoantibodies directed against K+ channels of peripheral nerves.
Paul Shillito;Peter C. Molenaar;Angela Vincent;Angela Vincent;Katherine Leys.
Annals of Neurology (1995)
Abnormal myotonic dystrophy protein kinase levels produce only mild myopathy in mice
Gert Jansen;P.J.T.A. Groenen;D. Bächner;P.H.K. Jap.
Nature Genetics (1996)
Monoclonal antibodies raised against Guillain-Barré syndrome–associated Campylobacter jejuni lipopolysaccharides react with neuronal gangliosides and paralyze muscle-nerve preparations
Carl S. Goodyear;Graham M. O’Hanlon;Jaap J. Plomp;Eric R. Wagner.
Journal of Clinical Investigation (1999)
Eculizumab prevents anti-ganglioside antibody-mediated neuropathy in a murine model.
Susan K. Halstead;Femke M. P. Zitman;Peter D. Humphreys;Kay Greenshields.
Miller Fisher anti-GQ1b antibodies: alpha-latrotoxin-like effects on motor end plates.
J. J. Plomp;P. C. Molenaar;G. M. O'hanlon;B. C. Jacobs.
Annals of Neurology (1999)
MuSK IgG4 autoantibodies cause myasthenia gravis by inhibiting binding between MuSK and Lrp4
Maartje G. Huijbers;Wei Zhang;Rinse Klooster;Erik H. Niks.
Proceedings of the National Academy of Sciences of the United States of America (2013)
Adaptation of quantal content to decreased postsynaptic sensitivity at single endplates in alpha-bungarotoxin-treated rats.
J. J. Plomp;G. T. H. Van Kempen;P. C. Molenaar.
The Journal of Physiology (1992)
Anti-GQ1b ganglioside antibodies mediate complement-dependent destruction of the motor nerve terminal
Graham M. O'Hanlon;Jaap J. Plomp;Mahua Chakrabarti;Ian Morrison.
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