D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 36 Citations 6,644 69 World Ranking 3965 National Ranking 109

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Neuron

Jaap J. Plomp mainly focuses on Neuromuscular junction, Internal medicine, Endocrinology, Neuroscience and Cell biology. His research integrates issues of Complement membrane attack complex, Acetylcholine and Motor nerve in his study of Neuromuscular junction. His work on Myocyte as part of his general Endocrinology study is frequently connected to Myotonin-protein kinase, thereby bridging the divide between different branches of science.

His research in Neuroscience intersects with topics in Missense mutation, Cortical spreading depression, Migraine and Neurotransmission. His Neurotransmission research incorporates elements of Synapse, Synaptic vesicle and Neurotransmitter. While the research belongs to areas of Cell biology, Jaap J. Plomp spends his time largely on the problem of Complement system, intersecting his research to questions surrounding Ganglioside.

His most cited work include:

  • Synaptic Assembly of the Brain in the Absence of Neurotransmitter Secretion (1055 citations)
  • A Cacna1a Knockin Migraine Mouse Model with Increased Susceptibility to Cortical Spreading Depression (536 citations)
  • Acquired neuromyotonia: evidence for autoantibodies directed against K+ channels of peripheral nerves. (317 citations)

What are the main themes of his work throughout his whole career to date?

Jaap J. Plomp mostly deals with Neuromuscular junction, Neuroscience, Acetylcholine, Internal medicine and Immunology. Jaap J. Plomp interconnects Postsynaptic potential, Neurotransmitter, Neurotransmission, Neuromuscular transmission and Acetylcholine receptor in the investigation of issues within Neuromuscular junction. His research investigates the connection between Neuromuscular transmission and topics such as Anatomy that intersect with problems in Synaptic vesicle.

His Neuroscience research integrates issues from Familial hemiplegic migraine and Cell biology. His studies deal with areas such as Endocrinology and Cardiology as well as Internal medicine. His Endocrinology study integrates concerns from other disciplines, such as Facial muscles and Receptor tyrosine kinase.

He most often published in these fields:

  • Neuromuscular junction (47.75%)
  • Neuroscience (41.44%)
  • Acetylcholine (27.03%)

What were the highlights of his more recent work (between 2017-2021)?

  • Myasthenia gravis (18.02%)
  • Acetylcholine receptor (20.72%)
  • Immunology (24.32%)

In recent papers he was focusing on the following fields of study:

Myasthenia gravis, Acetylcholine receptor, Immunology, Antibody and Neuromuscular junction are his primary areas of study. In his work, Phosphorylation, Molecular biology, Agrin and Affinity maturation is strongly intertwined with Monoclonal antibody, which is a subfield of Myasthenia gravis. Acetylcholine receptor is a subfield of Internal medicine that Jaap J. Plomp studies.

His Neuromuscular junction research incorporates themes from Postsynaptic potential, mdx mouse, Dystrophin, Muscle weakness and Neurotransmission. His Postsynaptic potential research is multidisciplinary, incorporating perspectives in Motor neuron, Skeletal muscle, Neuroscience, Neuromuscular transmission and Cell biology. He combines subjects such as Endocrinology, Facial weakness, Neonatal Fc receptor, Electromyography and In vivo with his study of Muscle weakness.

Between 2017 and 2021, his most popular works were:

  • MuSK myasthenia gravis monoclonal antibodies: Valency dictates pathogenicity. (29 citations)
  • Natural disease history of the D2-mdx mouse model for Duchenne muscular dystrophy (27 citations)
  • IgG4-mediated autoimmune diseases: a niche of antibody-mediated disorders. (26 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Neuron

Jaap J. Plomp mainly investigates Antibody, Antigen, Myasthenia gravis, Sinus rhythm and Inflammation. His Antibody research is multidisciplinary, relying on both Molecular biology, Phosphorylation and Agrin. As part of his studies on Antigen, Jaap J. Plomp often connects relevant subjects like Acetylcholine receptor.

His Sinus rhythm research includes elements of Right atrial and Arrhythmia detection. His Inflammation study combines topics from a wide range of disciplines, such as Autoantibody, Neuromuscular junction, Receptor and Autoimmunity. His Adenoassociated virus research includes a combination of various areas of study, such as Rhythm, Internal medicine and Atrial fibrillation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Synaptic Assembly of the Brain in the Absence of Neurotransmitter Secretion

M Verhage;A S Maia;J J Plomp;A B Brussaard.
Science (2000)

1373 Citations

A Cacna1a Knockin Migraine Mouse Model with Increased Susceptibility to Cortical Spreading Depression

Arn M.J.M van den Maagdenberg;Daniela Pietrobon;Tommaso Pizzorusso;Simon Kaja.
Neuron (2004)

712 Citations

Acquired neuromyotonia: evidence for autoantibodies directed against K+ channels of peripheral nerves.

Paul Shillito;Peter C. Molenaar;Angela Vincent;Angela Vincent;Katherine Leys.
Annals of Neurology (1995)

445 Citations

Abnormal myotonic dystrophy protein kinase levels produce only mild myopathy in mice

Gert Jansen;P.J.T.A. Groenen;D. Bächner;P.H.K. Jap.
Nature Genetics (1996)

365 Citations

Monoclonal antibodies raised against Guillain-Barré syndrome–associated Campylobacter jejuni lipopolysaccharides react with neuronal gangliosides and paralyze muscle-nerve preparations

Carl S. Goodyear;Graham M. O’Hanlon;Jaap J. Plomp;Eric R. Wagner.
Journal of Clinical Investigation (1999)

308 Citations

Eculizumab prevents anti-ganglioside antibody-mediated neuropathy in a murine model.

Susan K. Halstead;Femke M. P. Zitman;Peter D. Humphreys;Kay Greenshields.
Brain (2008)

249 Citations

Miller Fisher anti-GQ1b antibodies: alpha-latrotoxin-like effects on motor end plates.

J. J. Plomp;P. C. Molenaar;G. M. O'hanlon;B. C. Jacobs.
Annals of Neurology (1999)

220 Citations

MuSK IgG4 autoantibodies cause myasthenia gravis by inhibiting binding between MuSK and Lrp4

Maartje G. Huijbers;Wei Zhang;Rinse Klooster;Erik H. Niks.
Proceedings of the National Academy of Sciences of the United States of America (2013)

205 Citations

Adaptation of quantal content to decreased postsynaptic sensitivity at single endplates in alpha-bungarotoxin-treated rats.

J. J. Plomp;G. T. H. Van Kempen;P. C. Molenaar.
The Journal of Physiology (1992)

196 Citations

Anti-GQ1b ganglioside antibodies mediate complement-dependent destruction of the motor nerve terminal

Graham M. O'Hanlon;Jaap J. Plomp;Mahua Chakrabarti;Ian Morrison.
Brain (2001)

192 Citations

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