His primary areas of study are Genetics, Genome-wide association study, Schizophrenia, Bipolar disorder and Locus. His Genetics study often links to related topics such as Psychosis. His studies deal with areas such as Copy-number variation, Psychiatry, Case-control study, Linkage disequilibrium and Genetic association as well as Genome-wide association study.
His research integrates issues of Schizoaffective disorder, Genetic variation and ANK3 in his study of Genetic association. His work carried out in the field of Schizophrenia brings together such families of science as Major depressive disorder, Disease, Allele and Genetic architecture. His Locus research is multidisciplinary, incorporating perspectives in Founder effect and Spinocerebellar ataxia.
The scientist’s investigation covers issues in Genetics, Genome-wide association study, Gene, Schizophrenia and Genetic association. His study in Single-nucleotide polymorphism, Locus, Genetic linkage, Candidate gene and Haplotype is done as part of Genetics. The concepts of his Genetic linkage study are interwoven with issues in Genetic marker and Linkage.
His work in Genome-wide association study tackles topics such as Psychiatry which are related to areas like Neuroticism. Brien P. Riley works mostly in the field of Schizophrenia, limiting it down to concerns involving Psychosis and, occasionally, Genetic determinism. His Genetic association research incorporates themes from Odds ratio, Meta-analysis, Disease, Imputation and Summary statistics.
Brien P. Riley mainly focuses on Genome-wide association study, Gene, Genetics, Computational biology and Genetic association. His Genome-wide association study research is multidisciplinary, incorporating elements of Eating disorders and Psychiatry. His study ties his expertise on Schizophrenia together with the subject of Genetics.
His study in Computational biology is interdisciplinary in nature, drawing from both Cell, Gene expression, Expression data and Identity by descent. His Genetic association study combines topics from a wide range of disciplines, such as Odds ratio, DNA sequencing and Haplotype. Brien P. Riley has included themes like Schizophrenia, Biological pathway, Bipolar disorder, Neuropsychiatric disorder and De novo mutations in his Genetic architecture study.
Brien P. Riley focuses on Genetic architecture, Gene, Genome-wide association study, Schizophrenia and Psychiatry. His Genetic architecture study is concerned with Genetics in general. Brien P. Riley works in the field of Genetics, focusing on Phenotype in particular.
His work deals with themes such as Major histocompatibility complex, Genetic association and Depression, which intersect with Genome-wide association study. His Genetic association study combines topics in areas such as Gene expression, Haplotype and Human genome. His Schizophrenia research includes themes of Eating disorders, Anorexia nervosa, Clinical psychology and Binge eating.
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Biological insights from 108 schizophrenia-associated genetic loci
Stephan Ripke;Stephan Ripke;Benjamin M. Neale;Benjamin M. Neale;Aiden Corvin;James T. R. Walters.
Identification of risk loci with shared effects on five major psychiatric disorders: a genome-wide analysis
Jordan W. Smoller;Kenneth Kendler;Nicholas John Craddock;Phil Hyoun Lee.
The Lancet (2013)
Genome-wide association study identifies five new schizophrenia loci
Stephan Ripke;Alan R. Sanders;Kenneth S. Kendler;Douglas F. Levinson.
Nature Genetics (2011)
Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression
Naomi R. Wray;Stephan Ripke;Stephan Ripke;Stephan Ripke;Manuel Mattheisen;MacIej Trzaskowski.
Nature Genetics (2018)
Genome-wide association analysis identifies 13 new risk loci for schizophrenia
Stephan Ripke;Stephan Ripke;Colm T. O'Dushlaine;Kimberly D. Chambert;Jennifer L. Moran.
Nature Genetics (2013)
Modeling Linkage Disequilibrium Increases Accuracy of Polygenic Risk Scores
Bjarni J. Vilhjálmsson;Jian Yang;Hilary K. Finucane;Alexander Gusev.
American Journal of Human Genetics (2015)
Detection of an unstable fragment of DNA specific to individuals with myotonic dystrophy.
J Buxton;P Shelbourne;J Davies;C Jones.
Childhood adversity, monoamine oxidase a genotype, and risk for conduct disorder.
Debra L. Foley;Lindon J. Eaves;Brandon Wormley;Judy L. Silberg.
Archives of General Psychiatry (2004)
Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects
Christian R Marshall;Daniel P Howrigan;Daniel P Howrigan;Daniele Merico;Bhooma Thiruvahindrapuram.
Nature Genetics (2017)
Sparse whole-genome sequencing identifies two loci for major depressive disorder
Na Cai;Tim B. Bigdeli;Warren Kretzschmar;Yihan Li.
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