What is he best known for?
The fields of study he is best known for:
- Gene
- Neuron
- Neurotransmitter
Tomi Taira focuses on Neuroscience, Hippocampal formation, Long-term potentiation, Hippocampus and GABAA receptor.
The concepts of his Neuroscience study are interwoven with issues in Synaptic plasticity, Postsynaptic potential, Neurotransmission and Kainate receptor.
His biological study spans a wide range of topics, including Extracellular and Inhibitory postsynaptic potential.
The Long-term potentiation study combines topics in areas such as Signal transduction and Cell biology.
Basal, Downregulation and upregulation and Nervous system is closely connected to Gene targeting in his research, which is encompassed under the umbrella topic of Hippocampus.
His GABAA receptor research incorporates elements of Stimulation and Excitatory postsynaptic potential.
His most cited work include:
- Long-Lasting GABA-Mediated Depolarization Evoked by High-Frequency Stimulation in Pyramidal Neurons of Rat Hippocampal Slice Is Attributable to a Network-Driven, Bicarbonate-Dependent K+ Transient (313 citations)
- Transgenic mice overexpressing the full-length neurotrophin receptor trkB exhibit increased activation of the trkB–PLCγ pathway, reduced anxiety, and facilitated learning (147 citations)
- Syndecan-3-deficient mice exhibit enhanced LTP and impaired hippocampus-dependent memory (133 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Neuroscience, Hippocampus, Long-term potentiation, Neurotransmission and Hippocampal formation.
Tomi Taira combines subjects such as Synaptic plasticity, Postsynaptic potential, Glutamate receptor, Glutamatergic and Kainate receptor with his study of Neuroscience.
In his study, Stimulation, Monoamine neurotransmitter and Diazepam is strongly linked to GABAA receptor, which falls under the umbrella field of Hippocampus.
His research integrates issues of NMDA receptor, Gene targeting, Long-term depression and Tropomyosin receptor kinase B in his study of Long-term potentiation.
His studies deal with areas such as GABAergic, Giant depolarizing potentials and Synaptogenesis as well as Neurotransmission.
Tomi Taira has included themes like Extracellular, Inhibitory postsynaptic potential and Depolarization in his Hippocampal formation study.
He most often published in these fields:
- Neuroscience (64.55%)
- Hippocampus (31.82%)
- Long-term potentiation (25.45%)
What were the highlights of his more recent work (between 2016-2021)?
- Neuroscience (64.55%)
- Hippocampus (31.82%)
- Kainate receptor (18.18%)
In recent papers he was focusing on the following fields of study:
His main research concerns Neuroscience, Hippocampus, Kainate receptor, Long-term potentiation and Tropomyosin receptor kinase B.
His Neuroscience research includes elements of Glutamate receptor and Metaplasticity.
His studies in Hippocampus integrate themes in fields like Neurotrophic factors, Inhibitory postsynaptic potential and Neurotransmission.
His biological study deals with issues like Hippocampal formation, which deal with fields such as GABAergic.
Within one scientific family, he focuses on topics pertaining to Dendritic spine under Kainate receptor, and may sometimes address concerns connected to Protein subunit.
Tomi Taira interconnects Synaptic plasticity and Glutamatergic in the investigation of issues within Synaptogenesis.
Between 2016 and 2021, his most popular works were:
- Isoflurane produces antidepressant effects and induces TrkB signaling in rodents (41 citations)
- Chronic fluoxetine administration enhances synaptic plasticity and increases functional dynamics in hippocampal CA3-CA1 synapses. (23 citations)
- NETO1 Guides Development of Glutamatergic Connectivity in the Hippocampus by Regulating Axonal Kainate Receptors. (13 citations)
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