D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 54 Citations 13,389 116 World Ranking 10904 National Ranking 109

Overview

What is he best known for?

The fields of study Siegfried Waldegger is best known for:

  • Gene
  • Dopamine
  • Neurotransmitter

His study on Biochemistry is mostly dedicated to connecting different topics, such as Pi. Siegfried Waldegger brings together Cell biology and Molecular biology to produce work in his papers. Siegfried Waldegger applies his multidisciplinary studies on Molecular biology and Biochemistry in his research. As part of his studies on Receptor, Siegfried Waldegger often connects relevant subjects like Bradykinin. Siegfried Waldegger merges many fields, such as Biophysics and Cell biology, in his writings. His Endocrinology study frequently draws parallels with other fields, such as Kidney. Siegfried Waldegger combines topics linked to Endocrinology with his work on Kidney. Siegfried Waldegger performs integrative Gene and HEK 293 cells research in his work. His HEK 293 cells study typically links adjacent topics like Internal medicine.

His most cited work include:

  • Blockade of HERG channels expressed in Xenopus oocytes by the histamine receptor antagonists terfenadine and astemizole (217 citations)
  • Monoamine neurotransmitter transport mediated by the polyspecific cation transporter rOCT1 (97 citations)
  • Role of N-linked glycosylation in rat renal Na/Pi-cotransport. (94 citations)

What are the main themes of his work throughout his whole career to date

Siegfried Waldegger bridges between several scientific fields such as Kidney and Depolarization in his study of Endocrinology. Many of his studies involve connections with topics such as Endocrinology and Kidney. Depolarization is closely attributed to Biophysics in his work. Many of his studies on Biophysics apply to Potassium channel blocker as well. A component of his Sodium study involves Cotransporter and Epithelial sodium channel. Siegfried Waldegger integrates several fields in his works, including Epithelial sodium channel and Sodium. HERG and Potassium channel blocker are the subject areas of his Potassium channel study. In his works, Siegfried Waldegger performs multidisciplinary study on hERG and Potassium channel. His Biochemistry study frequently draws connections to adjacent fields such as Blockade.

Siegfried Waldegger most often published in these fields:

  • Biochemistry (91.67%)
  • Biophysics (54.17%)
  • Gene (54.17%)

What were the highlights of his more recent work (between 2000-2002)?

  • Gene (100.00%)
  • Biochemistry (100.00%)
  • Kinase (100.00%)

In recent works Siegfried Waldegger was focusing on the following fields of study:

His multidisciplinary approach integrates Gene and Intracellular in his work. His study deals with a combination of Intracellular and Transcription factor. His research links Biochemistry with Transcription factor. As part of his studies on Biochemistry, Siegfried Waldegger frequently links adjacent subjects like Protein kinase A. Siegfried Waldegger applies his multidisciplinary studies on Protein kinase A and Kinase in his research. As part of his studies on Kinase, Siegfried Waldegger frequently links adjacent subjects like SGK1. His research combines Endocrinology and SGK1. His studies link Aldosterone with Endocrinology. In his works, he conducts interdisciplinary research on Aldosterone and Epithelial sodium channel.

Between 2000 and 2002, his most popular works were:

  • Cerebral localization and regulation of the cell volume-sensitive serum- and glucocorticoid-dependent kinase SGK1 (79 citations)
  • Serum- and glucocorticoid-dependent kinase, cell volume, and the regulation of epithelial transport (22 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Functional Significance of Cell Volume Regulatory Mechanisms

Florian Lang;Gillian L. Busch;Markus Ritter;Harald Völkl.
Physiological Reviews (1998)

2154 Citations

Hypomagnesemia with secondary hypocalcemia is caused by mutations in TRPM6, a new member of the TRPM gene family.

Karl P Schlingmann;Stefanie Weber;Melanie Peters;Lene Niemann Nejsum.
Nature Genetics (2002)

846 Citations

A constitutively open potassium channel formed by KCNQ1 and KCNE3

Björn C. Schroeder;Siegfried Waldegger;Susanne Fehr;Markus Bleich.
Nature (2000)

654 Citations

Cloning and characterization of a putative human serine/threonine protein kinase transcriptionally modified during anisotonic and isotonic alterations of cell volume

Siegfried Waldegger;Petra Barth;Gertraud Raber;Florian Lang.
Proceedings of the National Academy of Sciences of the United States of America (1997)

563 Citations

Intracellular anions as the voltage sensor of prestin, the outer hair cell motor protein

Dominik Oliver;David Z. Z. He;Nikolaj Klöcker;Jost Ludwig.
Science (2001)

521 Citations

Mutations in the Tight-Junction Gene Claudin 19 (CLDN19) Are Associated with Renal Magnesium Wasting, Renal Failure, and Severe Ocular Involvement

Martin Konrad;André Schaller;Dominik Seelow;Amit V. Pandey.
American Journal of Human Genetics (2006)

477 Citations

Disruption of TRPM6/TRPM7 complex formation by a mutation in the TRPM6 gene causes hypomagnesemia with secondary hypocalcemia

Vladimir Chubanov;Siegfried Waldegger;Michael Mederos y Schnitzler;Helga Vitzthum.
Proceedings of the National Academy of Sciences of the United States of America (2004)

437 Citations

Claudin-16 and claudin-19 interact and form a cation-selective tight junction complex.

Jianghui Hou;Aparna Renigunta;Martin Konrad;Antonio S. Gomes.
Journal of Clinical Investigation (2008)

360 Citations

Blockade of HERG channels expressed in Xenopus oocytes by the histamine receptor antagonists terfenadine and astemizole

H. Suessbrich;S. Waldegger;F. Lang;A.E. Busch.
FEBS Letters (1996)

330 Citations

Salt Wasting and Deafness Resulting from Mutations in Two Chloride Channels

Karl P. Schlingmann;Martin Konrad;Nikola Jeck;Petra Waldegger.
The New England Journal of Medicine (2004)

307 Citations

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