D-Index & Metrics Best Publications
Neuroscience
Japan
2023

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 64 Citations 14,532 182 World Ranking 1909 National Ranking 51

Research.com Recognitions

Awards & Achievements

2023 - Research.com Neuroscience in Japan Leader Award

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • Internal medicine

Kohichi Tanaka focuses on Neuroscience, Glutamate receptor, Cell biology, Endocrinology and Internal medicine. Kohichi Tanaka has researched Neuroscience in several fields, including NMDA receptor, Metabotropic glutamate receptor 1, Metabotropic glutamate receptor, Nerve growth factor and Neurotrophic factors. His study brings together the fields of Neurotransmitter and Glutamate receptor.

His Cell biology research incorporates themes from Mutant, Gene, Astrocyte, Reticular connective tissue and Glial fibrillary acidic protein. His Excitotoxicity research is multidisciplinary, incorporating perspectives in Excitatory postsynaptic potential, Hippocampal formation, SLC1A2, Axon and Synapse. His studies deal with areas such as Glutamate homeostasis, Glutamate Plasma Membrane Transport Proteins, Glutamate reuptake, Glutamate dehydrogenase 1 and SLC1A1 as well as Glutamate aspartate transporter.

His most cited work include:

  • Epilepsy and Exacerbation of Brain Injury in Mice Lacking the Glutamate Transporter GLT-1 (1468 citations)
  • Structure and functional expression of the cloned rat neurotensin receptor (476 citations)
  • Molecular characterization of a functional cDNA for rat substance P receptor. (419 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Neuroscience, Glutamate receptor, Cell biology, Internal medicine and Endocrinology. In Neuroscience, he works on issues like Metabotropic glutamate receptor 1, which are connected to Metabotropic glutamate receptor 7. As a member of one scientific family, Kohichi Tanaka mostly works in the field of Glutamate receptor, focusing on Neurotransmission and, on occasion, Neurotransmitter.

His study looks at the intersection of Cell biology and topics like Purkinje cell with Synaptic cleft. Kohichi Tanaka focuses mostly in the field of Biochemistry, narrowing it down to topics relating to Retina and, in certain cases, Neuroprotection. His Glutamate aspartate transporter study incorporates themes from Complementary DNA and Gene expression.

He most often published in these fields:

  • Neuroscience (44.78%)
  • Glutamate receptor (42.29%)
  • Cell biology (32.84%)

What were the highlights of his more recent work (between 2013-2021)?

  • Neuroscience (44.78%)
  • Cell biology (32.84%)
  • Glutamate receptor (42.29%)

In recent papers he was focusing on the following fields of study:

Kohichi Tanaka mainly focuses on Neuroscience, Cell biology, Glutamate receptor, Gene and Computational biology. His Cell biology research is multidisciplinary, incorporating elements of Stimulation, Gene knockin and CRISPR. He studies Glutamate receptor, focusing on AMPA receptor in particular.

His Computational biology research is multidisciplinary, relying on both Genome editing and DNA. His research in Glutamic acid tackles topics such as Programmed cell death which are related to areas like Glutamate aspartate transporter. His NMDA receptor study combines topics from a wide range of disciplines, such as Synapse, Endocrinology and Receptor antagonist.

Between 2013 and 2021, his most popular works were:

  • Cloning-free CRISPR/Cas System Facilitates Functional Cassette Knock-In in Mice (189 citations)
  • Glial dysfunction in the mouse habenula causes depressive-like behaviors and sleep disturbance (82 citations)
  • Astroglial glutamate transporter deficiency increases synaptic excitability and leads to pathological repetitive behaviors in mice. (76 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • Internal medicine

His scientific interests lie mostly in Neuroscience, Glutamate receptor, Cell biology, Gene and Gene targeting. The study incorporates disciplines such as Electrophysiology, Neurotransmitter, Neuroprotection, Neurotransmission and Parallel fiber in addition to Glutamate receptor. His Neuroprotection research includes elements of Excitotoxicity, AMPA receptor, Motor neuron and Nuclear pore.

Cell biology is closely attributed to Cell growth in his work. His Gene targeting study deals with Gene knockin intersecting with Cloning, Cas9, Molecular biology, RNA and Guide RNA. As part of the same scientific family, Kohichi Tanaka usually focuses on Excitatory postsynaptic potential, concentrating on Neuron and intersecting with NMDA receptor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Epilepsy and Exacerbation of Brain Injury in Mice Lacking the Glutamate Transporter GLT-1

Kohichi Tanaka;Kei Watase;Toshiya Manabe;Keiko Yamada.
Science (1997)

1933 Citations

Structure and functional expression of the cloned rat neurotensin receptor

Kohichi Tanaka;Masayuki Masu;Shigetada Nakanishi.
Neuron (1990)

624 Citations

Molecular characterization of a functional cDNA for rat substance P receptor.

Y Yokota;Y Sasai;K Tanaka;T Fujiwara.
Journal of Biological Chemistry (1989)

577 Citations

Glutamate Transporter GLAST Is Expressed in the Radial Glia–Astrocyte Lineage of Developing Mouse Spinal Cord

Takashi Shibata;Keiko Yamada;Masahiko Watanabe;Kazuhiro Ikenaka.
The Journal of Neuroscience (1997)

465 Citations

Motor discoordination and increased susceptibility to cerebellar injury in GLAST mutant mice.

Kei Watase;Kouichi Hashimoto;Masanobu Kano;Keiko Yamada.
European Journal of Neuroscience (1998)

457 Citations

Microglia–Müller Glia Cell Interactions Control Neurotrophic Factor Production during Light-Induced Retinal Degeneration

Takayuki Harada;Chikako Harada;Shinichi Kohsaka;Etsuko Wada.
The Journal of Neuroscience (2002)

452 Citations

Inducible gene deletion in astroglia and radial glia- : A valuable tool for functional and lineage analysis

Tetsuji Mori;Kohichi Tanaka;Annalisa Buffo;Wolfgang Wurst.
Glia (2006)

392 Citations

Glial Glutamate Transporters Mediate a Functional Metabolic Crosstalk between Neurons and Astrocytes in the Mouse Developing Cortex

Brigitte Voutsinos-Porche;Gilles Bonvento;Kohichi Tanaka;Pascal Steiner.
Neuron (2003)

328 Citations

The potential role of glutamate transporters in the pathogenesis of normal tension glaucoma

Takayuki Harada;Chikako Harada;Kazuaki Nakamura;Hun-Meng A. Quah.
Journal of Clinical Investigation (2007)

326 Citations

The dorsomedial hypothalamic nucleus as a putative food-entrainable circadian pacemaker

Michihiro Mieda;S. Clay Williams;James A. Richardson;Kohichi Tanaka.
Proceedings of the National Academy of Sciences of the United States of America (2006)

326 Citations

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