His primary areas of study are Microbiome, Pregnancy, Metagenomics, Physiology and Immunology. Human Microbiome Project is the focus of his Microbiome research. His Metagenomics study combines topics from a wide range of disciplines, such as Oral Microbiome, Human microbiome and Pathology.
His Physiology research includes elements of Internal medicine and Obesity. His Immunology course of study focuses on Gestation and Gestational age, Placenta and Placental Membrane. His Meconium research integrates issues from Delivery mode, Gastrointestinal Microbiome, Cohort and Bioinformatics.
The scientist’s investigation covers issues in Microbiome, Physiology, Genetics, Immunology and Metagenomics. His studies in Microbiome integrate themes in fields like Offspring, Pregnancy, Chorioamnionitis, Gestation and Obstetrics. His study in the fields of Meconium under the domain of Pregnancy overlaps with other disciplines such as Reproductive health and Genome-wide association study.
His Physiology research is multidisciplinary, incorporating elements of Endocrinology, Obesity, Internal medicine, Intestinal Microbiome and Dysbiosis. His research in Genetics tackles topics such as Computational biology which are related to areas like RNA RIBOSOMAL 16S, Gene expression profiling, Cloning, Promoter and Proteomics. Jun Ma combines subjects such as Prospective cohort study, Human microbiome and Bioinformatics with his study of Metagenomics.
His primary areas of study are Microbiome, Physiology, Gestation, Fetus and Chorioamnionitis. His study on Microbiome also encompasses disciplines like
His Dysbiosis research focuses on subjects like Feces, which are linked to Gastrointestinal Microbiome. The concepts of his Chorioamnionitis study are interwoven with issues in Betamethasone and Placenta, Placental Membrane. The study incorporates disciplines such as Pregnancy, Prospective cohort study and Meconium in addition to Obesity.
Jun Ma mostly deals with Microbiome, Physiology, Fetus, Immunology and Gestation. In the field of Microbiome, his study on Gastrointestinal Microbiome overlaps with subjects such as Diversity. His work carried out in the field of Gastrointestinal Microbiome brings together such families of science as Offspring, Blood lipids, Cholesterol and Feces.
His Physiology study frequently involves adjacent topics like Dysbiosis. His studies examine the connections between Prospective cohort study and genetics, as well as such issues in Pregnancy, with regards to Metagenomics. His Chorioamnionitis study combines topics from a wide range of disciplines, such as Placenta, Placental Membrane and Premature birth.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The Placenta Harbors a Unique Microbiome
Kjersti Aagaard;Jun Ma;Kathleen M. Antony;Radhika Ganu.
Science Translational Medicine (2014)
Use of Arsenic Trioxide (As2O3 ) in the Treatment of Acute Promyelocytic Leukemia (APL): II. Clinical Efficacy and Pharmacokinetics in Relapsed Patients
Zhi-Xiang Shen;Guo-Qiang Chen;Jian-Hua Ni;Xiu-Shong Li.
Blood (1997)
GAL4-VP16 is an unusually potent transcriptional activator
Ivan Sadowski;Jun Ma;Steve Triezenberg;Mark Ptashne.
Nature (1988)
The genome of the social amoeba Dictyostelium discoideum
L. Eichinger;J. A. Pachebat;J. A. Pachebat;G. Glöckner;M.-A. Rajandream.
Nature (2005)
Deletion analysis of GAL4 defines two transcriptional activating segments
Jun Ma;Mark Ptashne.
Cell (1987)
Soft Combination and Detection for Cooperative Spectrum Sensing in Cognitive Radio Networks
Jun Ma;Guodong Zhao;Ye Li.
IEEE Transactions on Wireless Communications (2008)
Soft Combination and Detection for Cooperative Spectrum Sensing in Cognitive Radio Networks
Jun Ma;Ye Li.
global communications conference (2007)
In vitro studies on cellular and molecular mechanisms of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia: As2O3 induces NB4 cell apoptosis with downregulation of Bcl-2 expression and modulation of PML-RAR alpha/PML proteins
Guo-Qiang Chen;Jun Zhu;Xue-Geng Shi;Jian-Hua Ni.
Blood (1996)
A new class of yeast transcriptional activators.
Jun Ma;Mark Ptashne.
Cell (1987)
Use of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia (APL) : I. As2O3 exerts dose-dependent dual effects on APL cells
Guo-Qiang Chen;Xue-Geng Shi;Wei Tang;Shu-Min Xiong.
Blood (1997)
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