What is he best known for?
The fields of study he is best known for:
- Gene
- Neurotransmitter
- Genetics
J. Troy Littleton spends much of his time researching Cell biology, Neurotransmitter, Neurotransmission, Vesicle fusion and Synaptotagmin 1.
He combines Cell biology and Dorsal closure in his studies.
He usually deals with Neurotransmitter and limits it to topics linked to Neuroscience and Receptor, VAMP2 and Complexin.
His study looks at the intersection of Neurotransmission and topics like Synaptic vesicle with Postsynaptic potential.
His Synaptotagmin 1 research focuses on Synaptotagmin I and how it relates to Biochemistry and Synaptotagmins.
His studies deal with areas such as SNAPAP and SNARE complex as well as STX1A.
His most cited work include:
- Insights into social insects from the genome of the honeybee Apis mellifera (1472 citations)
- Comparative Genomics of the Eukaryotes (1459 citations)
- Ion channels and synaptic organization: analysis of the Drosophila genome. (403 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Cell biology, Synaptic vesicle, Synaptotagmin 1, Neuroscience and Vesicle fusion.
His Cell biology research includes elements of STX1A, Postsynaptic potential, Neurotransmitter, Neurotransmission and Complexin.
His studies in Synaptic vesicle integrate themes in fields like Synapse, SNARE complex, Lipid bilayer fusion and Syntaxin.
His Synaptotagmin 1 research incorporates themes from Synaptic vesicle exocytosis, Synaptic augmentation, Synaptotagmin I and Synaptotagmins.
His studies deal with areas such as Synaptic plasticity and Endocytosis as well as Neuroscience.
His Vesicle fusion research includes themes of SNAP25, SNARE binding, Cooperativity and Kiss-and-run fusion.
He most often published in these fields:
- Cell biology (78.91%)
- Synaptic vesicle (37.50%)
- Synaptotagmin 1 (36.72%)
What were the highlights of his more recent work (between 2017-2021)?
- Neuroscience (29.69%)
- Cell biology (78.91%)
- Synaptic plasticity (17.97%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Neuroscience, Cell biology, Synaptic plasticity, Postsynaptic potential and Synaptotagmin 1.
The Neuroscience study combines topics in areas such as Active zone, Endocytosis, Gene knockdown and Endosome.
His Cell biology study incorporates themes from Cortex and Drosophila.
He interconnects Tonic, Neurotransmission, Neurotransmitter and Neuron in the investigation of issues within Synaptic plasticity.
His Neurotransmission study combines topics from a wide range of disciplines, such as Neuromuscular junction and Interneuron.
His Synaptotagmin 1 study introduces a deeper knowledge of Synaptic vesicle.
Between 2017 and 2021, his most popular works were:
- Characterization of developmental and molecular factors underlying release heterogeneity at Drosophila synapses (39 citations)
- Glial Ca2+signaling links endocytosis to K+ buffering around neuronal somas to regulate excitability (15 citations)
- Identification of a new SYT2 variant validates an unusual distal motor neuropathy phenotype. (9 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Genetics
- Neurotransmitter
J. Troy Littleton mainly focuses on Neuroscience, Synaptic plasticity, Cell biology, Neurotransmitter and Synapse.
His Neuroscience research is multidisciplinary, incorporating elements of Internalization, Endocytosis, Endosome and Calcium signaling.
The concepts of his Synaptic plasticity study are interwoven with issues in Active zone, Tonic, Postsynaptic potential and Neuron.
His research integrates issues of Glutamatergic, Homeostatic plasticity, Premovement neuronal activity, Neuronal circuits and Neuroplasticity in his study of Postsynaptic potential.
His Cell biology research is multidisciplinary, relying on both SYT7, Drosophila and Synaptotagmin 1.
His Neurotransmitter study integrates concerns from other disciplines, such as Motor neuron, Synaptic vesicle and Calcium imaging.
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