The scientist’s investigation covers issues in Cell biology, Osmotic shock, Mitogen-activated protein kinase, Cellular stress response and Regulation of gene expression. Dietmar Kültz does research in Cell biology, focusing on Signal transduction specifically. His study on Osmotic shock also encompasses disciplines like
The Mitogen-activated protein kinase study combines topics in areas such as c-Raf and Protein kinase A, p38 mitogen-activated protein kinases. His work carried out in the field of Cellular stress response brings together such families of science as Proteome, Stressor and Homeostasis. His Regulation of gene expression research incorporates elements of Gene expression and Osmolyte.
Dietmar Kültz mostly deals with Cell biology, Osmotic shock, Biochemistry, Oreochromis mossambicus and Proteome. His study in Cell biology is interdisciplinary in nature, drawing from both Osmotic concentration, Internal medicine, Endocrinology and Euryhaline. The concepts of his Osmotic shock study are interwoven with issues in DNA damage, Apoptosis, Transcription factor, Intracellular and Kidney.
As a part of the same scientific family, Dietmar Kültz mostly works in the field of Biochemistry, focusing on Osmoregulation and, on occasion, Function and Enhancer. His Proteome course of study focuses on Proteomics and Cytoskeleton and Fish Proteins. His work deals with themes such as Regulation of gene expression and Gene expression, which intersect with Osmolyte.
His primary areas of investigation include Cell biology, Proteome, Tilapia, Cellular stress response and Oreochromis mossambicus. His Cell biology research is multidisciplinary, relying on both Cell culture, CRISPR, Euryhaline, Renal physiology and Intron. His Proteome study combines topics from a wide range of disciplines, such as Proteomics, Gasterosteus, KEGG, Proteostasis and Gill.
His Tilapia research incorporates themes from Oreochromis, Data-independent acquisition, Promoter, Gene and Computational biology. His Cellular stress response research integrates issues from Comparative biology, Corporate social responsibility, Three-domain system and Bioinformatics. His Oreochromis mossambicus research is multidisciplinary, incorporating elements of Enhancer, Osmoregulation, Regulatory sequence and Osmotic concentration.
His primary areas of study are Cellular stress response, Ecology, Carbapenem-resistant enterobacteriaceae, Antibiotics and Relebactam. Much of his study explores Cellular stress response relationship to Systems biology. Dietmar Kültz combines topics linked to Osmotic shock with his work on Ecology.
His research in Carbapenem-resistant enterobacteriaceae intersects with topics in Vaborbactam and Avibactam.
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Molecular and evolutionary basis of the cellular stress response
Annual Review of Physiology (2005)
The ecoresponsive genome of Daphnia pulex
John K. Colbourne;Michael E. Pfrender;Michael E. Pfrender;Donald Gilbert;W. Kelley Thomas.
Regulation of gene expression by hypertonicity.
Maurice B. Burg;Eugene D. Kwon;Dietmar KÜltz.
Annual Review of Physiology (1997)
Evolution of the cellular stress proteome: from monophyletic origin to ubiquitous function.
The Journal of Experimental Biology (2003)
In vitro biologic activities of the antimicrobials triclocarban, its analogs, and triclosan in bioassay screens: receptor-based bioassay screens.
Ki Chang Ahn;Bin Zhao;Jiangang Chen;Gennady Cherednichenko.
Environmental Health Perspectives (2008)
Pathway analysis of kidney cancer using proteomics and metabolic profiling
Bertrand Perroud;Jinoo Lee;Nelly Valkova;Amy Dhirapong.
Molecular Cancer (2006)
Phylogenetic and Functional Classification of Mitogen- and Stress-Activated Protein Kinases
Journal of Molecular Evolution (1998)
Hyperosmolality Causes Growth Arrest of Murine Kidney Cells INDUCTION OF GADD45 AND GADD153 BY OSMOSENSING VIA STRESS-ACTIVATED PROTEIN KINASE 2
Dietmar Kültz;Samira Madhany;Maurice B. Burg.
Journal of Biological Chemistry (1998)
Evolution of osmotic stress signaling via MAP kinase cascades.
D Kültz;M Burg.
The Journal of Experimental Biology (1998)
Hyperosmolality in the form of elevated NaCl but not urea causes DNA damage in murine kidney cells
Dietmar Kültz;Devulapalli Chakravarty.
Proceedings of the National Academy of Sciences of the United States of America (2001)
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