Martha U. Gillette mainly investigates Suprachiasmatic nucleus, Circadian rhythm, Internal medicine, Endocrinology and Circadian clock. Her research in Suprachiasmatic nucleus intersects with topics in Glutamate receptor, Timeless and Cell biology. Many of her research projects under Circadian rhythm are closely connected to Clockwork with Clockwork, tying the diverse disciplines of science together.
She works mostly in the field of Light effects on circadian rhythm, limiting it down to topics relating to Period and, in certain cases, Endogeny. Her study in Hypothalamus and Retinohypothalamic tract falls within the category of Internal medicine. The study of Circadian clock is intertwined with the study of Melatonin in a number of ways.
Martha U. Gillette mainly focuses on Suprachiasmatic nucleus, Circadian rhythm, Circadian clock, Neuroscience and Internal medicine. Her Suprachiasmatic nucleus research is multidisciplinary, incorporating elements of Melatonin, Glutamate receptor, Premovement neuronal activity and Cell biology. She usually deals with Cell biology and limits it to topics linked to Genetics and Timeless.
Her research in Circadian rhythm tackles topics such as Slice preparation which are related to areas like Neuropeptide Y receptor. Her work in Circadian clock addresses subjects such as Neuropeptide, which are connected to disciplines such as Peptide. Her Endocrinology research extends to Internal medicine, which is thematically connected.
Her primary areas of investigation include Biophysics, Cell biology, Suprachiasmatic nucleus, Nanotechnology and Optics. Her biological study spans a wide range of topics, including Neurite growth, Premovement neuronal activity, Confluency, Neural engineering and Intracellular. Martha U. Gillette has researched Cell biology in several fields, including Metabotropic glutamate receptor 6, Metabotropic glutamate receptor 5, Inhibitory postsynaptic potential and Metabotropic glutamate receptor 7.
Circadian rhythm and Internal medicine are inextricably linked to her Suprachiasmatic nucleus research. Her Circadian rhythm study which covers Period that intersects with Glutamate receptor. Her work in Retinohypothalamic tract covers topics such as Pituitary adenylate cyclase-activating peptide which are related to areas like Endocrinology.
Her main research concerns Neuroscience, Suprachiasmatic nucleus, CLOCK, White paper and Ethical issues. Her study in the field of Reinnervation also crosses realms of New materials, Neuronal damage and Neuroblast. Her Suprachiasmatic nucleus research is under the purview of Circadian rhythm.
Her CLOCK study integrates concerns from other disciplines, such as Melatonin, Cell biology, Period and Zeitgeber. Her White paper research spans across into fields like Risk analysis, Induced pluripotent stem cell and Living systems.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Resetting the biological clock: mediation of nocturnal circadian shifts by glutamate and NO
JM Ding;D Chen;ET Weber;LE Faiman.
Spatial light interference microscopy (SLIM)
Zhuo Wang;Larry Millet;Mustafa Mir;Huafeng Ding.
Optics Express (2011)
Melatonin directly resets the rat suprachiasmatic circadian clock in vitro.
Angela J. McArthur;Martha U. Gillette;Rebecca A. Prosser.
Brain Research (1991)
Activation of MT2 melatonin receptors in rat suprachiasmatic nucleus phase advances the circadian clock
Amanda E. Hunt;Walid M. Al-Ghoul;Martha U. Gillette;Margarita L. Dubocovich.
American Journal of Physiology-cell Physiology (2001)
Pituitary Adenylate Cyclase-Activating Peptide (PACAP) in the Retinohypothalamic Tract: A Potential Daytime Regulator of the Biological Clock
Jens Hannibal;Jian M. Ding;Dong Chen;Jan Fahrenkrug.
The Journal of Neuroscience (1997)
Melatonin action and signal transduction in the rat suprachiasmatic circadian clock : Activation of protein kinase C at dusk and dawn
Angela J. McArthur;Amanda E. Hunt;Martha U. Gillette.
Ca2+/cAMP response element-binding protein (CREB)-dependent activation of Per1 is required for light-induced signaling in the suprachiasmatic nucleus circadian clock.
Shelley A. Tischkau;Shelley A. Tischkau;Jennifer W. Mitchell;Sheue Houy Tyan;Sheue Houy Tyan;Gordon F. Buchanan.
Journal of Biological Chemistry (2003)
Melatonin, sleep, and circadian rhythms: rationale for development of specific melatonin agonists
Fred W. Turek;Martha U. Gillette.
Sleep Medicine (2004)
Resetting the Biological Clock: Mediation of Nocturnal CREB Phosphorylation via Light, Glutamate, and Nitric Oxide
Jian M. Ding;Lia E. Faiman;William J. Hurst;Liana R. Kuriashkina.
The Journal of Neuroscience (1997)
A neuronal ryanodine receptor mediates light-induced phase delays of the circadian clock
Jian M. Ding;Gordon F. Buchanan;Shelley A. Tischkau;Dong Chen.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: