The scientist’s investigation covers issues in Toxicity, Pathology, Toxicology, Ecology and Anatomy. David E. Hinton has researched Toxicity in several fields, including Endocrinology, Yolk and Oryzias. His Endocrinology research is multidisciplinary, incorporating elements of Vitellogenin and Dipeptidyl peptidase.
His work carried out in the field of Pathology brings together such families of science as Carcinogenesis, Cytoplasm and Hepatocyte. The study incorporates disciplines such as Hazard, Diazinon and Environmental impact assessment in addition to Toxicology. His work on Ecotoxicology, Water pollution, Bioindicator and Contamination as part of general Ecology study is frequently linked to Environmental pollution, therefore connecting diverse disciplines of science.
David E. Hinton mainly investigates Oryzias, Toxicity, Internal medicine, Endocrinology and Molecular biology. His work deals with themes such as Andrology, Hatching, In vivo and Embryogenesis, which intersect with Oryzias. His Toxicity research is multidisciplinary, relying on both Environmental chemistry, Toxicology, Larva and Embryo.
The Internal medicine study combines topics in areas such as Vitellogenin and Cell biology. In his research on the topic of Endocrinology, Salmo is strongly related with Rainbow trout. Trout, Pathology, Cytoplasm, Bone canaliculus and Cell type is closely connected to Hepatocyte in his research, which is encompassed under the umbrella topic of Molecular biology.
His main research concerns Toxicity, Oryzias, Andrology, Embryo and Endocrinology. His study in Toxicity is interdisciplinary in nature, drawing from both Zoology, Fundulus, Juvenile fish and Environmental chemistry. His studies in Oryzias integrate themes in fields like Microscopy, Toxicology, DNA damage and Gill.
The various areas that David E. Hinton examines in his Embryo study include Organism, Developmental biology, Programmed cell death and Anatomy. The study incorporates disciplines such as Internal medicine, Sex reversal and Cell biology in addition to Endocrinology. David E. Hinton interconnects Microarray and Germ cell in the investigation of issues within Internal medicine.
His primary areas of study are Toxicity, Internal medicine, Endocrinology, Toxicology and Oryzias. His work on Acute toxicity as part of his general Toxicity study is frequently connected to Silver nanoparticle, thereby bridging the divide between different branches of science. His Internal medicine study combines topics in areas such as Cartilage and Cell biology.
David E. Hinton combines topics linked to Morphogenesis with his work on Endocrinology. His work carried out in the field of Toxicology brings together such families of science as Freshwater fish, Animal testing and Fish species. Within one scientific family, David E. Hinton focuses on topics pertaining to Embryo under Oryzias, and may sometimes address concerns connected to Gene expression.
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The Toxicology of Fishes
Richard T. Di Giulio;David E. Hinton.
(2008)
Mouse liver cell culture. I. Hepatocyte isolation.
James E. Klaunig;Peter J. Goldblatt;David E. Hinton;Michael M. Lipsky.
In Vitro Cellular & Developmental Biology – Plant (1981)
The fish embryo toxicity test as an animal alternative method in hazard and risk assessment and scientific research.
Michelle R. Embry;Scott E. Belanger;Thomas A. Braunbeck;Malyka Galay-Burgos.
Aquatic Toxicology (2010)
Histopathologic biomarkers in feral freshwater fish populations exposed to different types of contaminant stress
Swee J. Teh;S.M. Adams;David E. Hinton.
Aquatic Toxicology (1997)
Liver structural alterations accompanying chronic toxicity in fishes: Potential biomarkers of exposure
D.E. Hinton;D.J. Lauren;T.L. Holliday;C.S. Giam.
Preprints of Papers Presented at National Meeting, Division of Water, Air and Waste Chemistry, American Chemical Society; (USA) (1988)
Selenium biotransformations into proteinaceous forms by foodweb organisms of selenium-laden drainage waters in California.
Teresa W.-M Fan;Swee J Teh;David E Hinton;Richard M Higashi.
Aquatic Toxicology (2002)
Ascites, premature emergence, increased gonadal cell apoptosis, and cytochrome P4501A induction in pink salmon larvae continuously exposed to oil-contaminated gravel during development
Gary D. Marty;David E. Hinton;Jeffrey W. Short;Ronald A. Heintz.
Canadian Journal of Zoology (1997)
The use of bioindicators for assessing the effects of pollutant stress on fish
S.M. Adams;K.L. Shepard;M.S. Greeley;B.D. Jimenez.
Marine Environmental Research (1989)
In vitro modulation of 17-β-estradiol-induced vitellogenin synthesis: Effects of cytochrome P4501A1 inducing compounds on rainbow trout (Oncorhynchus mykiss) liver cells
Michael J. Anderson;Michael R. Miller;David E. Hinton.
Aquatic Toxicology (1996)
Diazinon and chlorpyrifos in urban waterways in northern California, USA.
Howard C. Bailey;Linda Deanovic;Emilie Reyes;Tom Kimball.
Environmental Toxicology and Chemistry (2000)
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