His primary areas of study are Genome-wide association study, Genetics, Single-nucleotide polymorphism, Schizophrenia and Bioinformatics. His Genome-wide association study research includes elements of Linkage disequilibrium, Genomics, Allele, Genetic association and Genetic architecture. His studies in Genetic association integrate themes in fields like Case-control study and Type 2 diabetes.
His work on Epigenetics, Human genetics and CpG site as part of general Genetics research is frequently linked to Cell aging and Induced pluripotent stem cell, thereby connecting diverse disciplines of science. His Single-nucleotide polymorphism research incorporates themes from Apolipoprotein E and Disease. The study incorporates disciplines such as Bipolar disorder, Psychosis, Brain mapping and Attention deficit hyperactivity disorder in addition to Schizophrenia.
His main research concerns Genetics, Genome-wide association study, Bipolar disorder, Single-nucleotide polymorphism and Schizophrenia. His study in Genetics concentrates on Locus, Gene, Genetic variation, Genetic determinism and Genomics. His work carried out in the field of Genome-wide association study brings together such families of science as Odds ratio, Bioinformatics, Case-control study, Allele and Genetic association.
Thomas W. Mühleisen focuses mostly in the field of Bipolar disorder, narrowing it down to matters related to Clinical psychology and, in some cases, Phenotype. His Single-nucleotide polymorphism study incorporates themes from Disease, Genetic predisposition and Haplotype. His Schizophrenia research is multidisciplinary, incorporating perspectives in Psychosis, Neuroscience, Brain mapping and Internal medicine.
His primary areas of investigation include Genome-wide association study, Genetics, Bipolar disorder, Computational biology and Schizophrenia. Thomas W. Mühleisen regularly links together related areas like Heritability in his Genome-wide association study studies. His Heritability research is multidisciplinary, incorporating elements of Single-nucleotide polymorphism, 1000 Genomes Project, Genetic correlation and Brain size.
His Genetics research focuses on Genotype-Phenotype Association in particular. His research investigates the connection with Computational biology and areas like Multiple comparisons problem which intersect with concerns in Genetic association, Genotype, Neurodevelopmental disorder and RNA. His Schizophrenia research includes themes of Copy-number variation, Multifactorial Inheritance, Psychosis and Disease, Age of onset.
Thomas W. Mühleisen mainly focuses on Schizophrenia, Gene, Genetic association, Bipolar disorder and Genome-wide association study. Thomas W. Mühleisen has researched Schizophrenia in several fields, including Genetics, Atrophy, Brain morphometry, Human brain and Depression. His study in Gene is interdisciplinary in nature, drawing from both Evolutionary biology and Multiple comparisons problem.
His Genetic association research includes elements of Neurodevelopmental disorder, Computational biology and Neuroscience. His research in Bipolar disorder intersects with topics in Copy-number variation, Multifactorial Inheritance, Psychosis and Disease, Age of onset.
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Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs
S. Hong Lee;Stephan Ripke;Stephan Ripke;Benjamin M. Neale;Benjamin M. Neale;Stephen V. Faraone.
Nature Genetics (2013)
Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease
Heribert Schunkert;Inke R. König;Sekar Kathiresan;Muredach P. Reilly.
Nature Genetics (2011)
Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes
Andrew P Morris;Benjamin F Voight;Benjamin F Voight;Tanya M Teslovich;Teresa Ferreira.
Nature Genetics (2012)
Large recurrent microdeletions associated with schizophrenia
Hreinn Stefansson;Dan Rujescu;Sven Cichon;Olli P. H. Pietilainen.
Nature (2008)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.
Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)
Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4
Pamela Sklar;Pamela Sklar;Stephan Ripke;Stephan Ripke;Laura J. Scott;Ole A. Andreassen.
Nature Genetics (2011)
Genetic associations with valvular calcification and aortic stenosis.
George Thanassoulis;George Thanassoulis;Catherine Y. Campbell;David S. Owens;J. Gustav Smith;J. Gustav Smith.
The New England Journal of Medicine (2013)
Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease.
Lesley Jones;Peter A. Holmans;Marian L. Hamshere;Denise Harold.
PLOS ONE (2010)
Common genetic variants influence human subcortical brain structures.
Derrek P. Hibar;Jason L. Stein;Jason L. Stein;Miguel E. Renteria;Alejandro Arias-Vasquez.
Nature (2015)
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