What is he best known for?
The fields of study he is best known for:
Michael R. Altherr mainly investigates Genetics, Gene, Locus, Chromosome 4 and Wolf–Hirschhorn syndrome.
His Genetics research is multidisciplinary, relying on both Molecular biology and Huntington's disease.
Michael R. Altherr has included themes like Dynamic mutation, Allele and Trinucleotide repeat expansion in his Huntington's disease study.
His Locus study incorporates themes from Constitutive heterochromatin, Human genome, Sequence analysis and Position-effect variegation.
Michael R. Altherr combines subjects such as Chromosome 21, Chromosome 3, Chromosome 17, Chromosome 16 and Chromosome 22 with his study of Chromosome 4.
The Wolf–Hirschhorn syndrome study combines topics in areas such as Phenotype, Fluorescence in situ hybridization and Gene mapping.
His most cited work include:
- A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes (6309 citations)
- Genomics of the Bacillus cereus group of organisms (391 citations)
- The DNA sequence and comparative analysis of human chromosome 20. (309 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Genetics, Gene, Chromosome 4, Molecular biology and Gene mapping.
His work in Locus, Wolf–Hirschhorn syndrome, Facioscapulohumeral muscular dystrophy, Genetic marker and Human genome are all subfields of Genetics research.
The concepts of his Locus study are interwoven with issues in Huntington's disease, Yeast artificial chromosome and Genomic library.
His Chromosome 4 research incorporates elements of Gene cluster, Gene expression and Chromosome 17.
His Molecular biology research is multidisciplinary, incorporating elements of Complementary DNA, FGF10 and Exon.
Michael R. Altherr interconnects Gene mutation, Cosmid and Breakpoint in the investigation of issues within Gene mapping.
He most often published in these fields:
- Genetics (113.75%)
- Gene (50.00%)
- Chromosome 4 (41.25%)
What were the highlights of his more recent work (between 2003-2008)?
- Genetics (113.75%)
- Sequence analysis (16.25%)
- Bacillus thuringiensis (7.50%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Genetics, Sequence analysis, Bacillus thuringiensis, Gene and Chromosome 16.
His Genetics study is mostly concerned with Gene duplication, Pseudogene, Segmental duplication, Whole genome sequencing and Chromosome 19.
His Gene duplication study integrates concerns from other disciplines, such as Physical Chromosome Mapping, Human genome, Locus and Chromosome 20.
His study in Chromosome 19 is interdisciplinary in nature, drawing from both Chromosome 4, Complete sequence, Chromosome 3, Chromosome 22 and Chromosome 7.
Michael R. Altherr focuses mostly in the field of Sequence analysis, narrowing it down to matters related to Microbiology and, in some cases, Virulence.
Gene family and Autosome are the subjects of his Gene studies.
Between 2003 and 2008, his most popular works were:
- Genomics of the Bacillus cereus group of organisms (391 citations)
- The DNA sequence and comparative analysis of human chromosome 20. (309 citations)
- The DNA sequence and comparative analysis of human chromosome 20. (309 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Genetics, Chromosome 22, Chromosome 16, Chromosome 17 and Chromosome 3.
Sequence analysis and Plasmid are subfields of Genetics in which his conducts study.
His biological study spans a wide range of topics, including Gene duplication, Chromosome 20, Pseudogene, Locus and Physical Chromosome Mapping.
The study incorporates disciplines such as Human genome and Segmental duplication in addition to Physical Chromosome Mapping.
His work deals with themes such as DNA microarray and Genome, Synteny, Genomics, which intersect with Plasmid.
His research integrates issues of Chromosome 4, Chromosome 21, Chromosome 19 and Chromosome 15 in his study of Chromosome 22.
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