University of Kentucky
His scientific interests lie mostly in Internal medicine, Endocrinology, Neurotoxicity, Pharmacology and Nicotine. His primary area of study in Endocrinology is in the field of Amygdala. His studies deal with areas such as Hippocampal formation, Glutamate receptor, Dopaminergic and NMDA receptor as well as Neurotoxicity.
The concepts of his Hippocampal formation study are interwoven with issues in Excitotoxicity and Cholinesterase. In his study, Acetylcholine receptor and Nicotinic acetylcholine receptor is inextricably linked to Antagonist, which falls within the broad field of Pharmacology. His Nicotine research incorporates elements of Anesthesia, Smoking cessation, Nicotinic agonist and Drug.
Mark A. Prendergast mainly focuses on Endocrinology, Internal medicine, Pharmacology, Neurotoxicity and NMDA receptor. Mark A. Prendergast studied Internal medicine and Ethanol that intersect with Alcohol. His work carried out in the field of Pharmacology brings together such families of science as Receptor, Antagonist, Nicotinic agonist, Nicotinic acetylcholine receptor and Nicotine.
The study incorporates disciplines such as Dizocilpine, Methamphetamine, Immunology and Caffeine in addition to Neurotoxicity. His studies in Immunology integrate themes in fields like Dopaminergic, Neuropathogenesis and Heroin. His research integrates issues of Glutamate receptor and Long-term potentiation in his study of NMDA receptor.
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Antagonist, Nicotine and Corticosterone. In most of his Internal medicine studies, his work intersects topics such as Neuron. His study in Endocrinology is interdisciplinary in nature, drawing from both NeuN and Receptor.
He has included themes like Pharmacology and Glucocorticoid in his Antagonist study. His Nicotine research focuses on subjects like Alcohol, which are linked to Naltrexone and Ethanol. Hippocampal formation is closely attributed to Neurotoxicity in his work.
Mark A. Prendergast spends much of his time researching Biochemistry, Antagonist, Internal medicine, Endocrinology and Stereochemistry. His Biochemistry research is multidisciplinary, incorporating perspectives in Neurotoxicity and Hippocampus. His Antagonist research is multidisciplinary, incorporating elements of Varenicline, Smoking cessation, Corticosterone and Pharmacology.
His research on Internal medicine often connects related areas such as Craving. Endocrinology is closely attributed to Neuron in his work. His research in the fields of Glycoside overlaps with other disciplines such as Streptomyces, Hexuronic Acids, Rare sugar and Terphenyl Compounds.
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Functional characterization of the novel neuronal nicotinic acetylcholine receptor ligand GTS-21 in vitro and in vivo.
Clark A Briggs;David J Anderson;Jorge D Brioni;Jerry J Buccafusco;Jerry J Buccafusco.
Pharmacology, Biochemistry and Behavior (1997)
Molecular basis for interactions of HIV and drugs of abuse.
Avi Nath;Kurt F. Hauser;Kurt F. Hauser;Valerie Wojna;Valerie Wojna;Rosemarie M. Booze;Rosemarie M. Booze.
Journal of Acquired Immune Deficiency Syndromes (2002)
Acamprosate Inhibits the Binding and Neurotoxic Effects of Trans‐ACPD, Suggesting a Novel Site of Action at Metabotropic Glutamate Receptors
Barton R. Harris;Mark A. Prendergast;D. Alex Gibson;D. Trent Rogers.
Alcoholism: Clinical and Experimental Research (2002)
Lesions of the nucleus paragigantocellularis: effects on mating behavior in male rats.
David P. Yells;David P. Yells;Shelton E. Hendricks;Shelton E. Hendricks;Mark A. Prendergast;Mark A. Prendergast.
Brain Research (1992)
Central nicotinic receptor agonists ABT-418, ABT-089, and (-)-nicotine reduce distractibility in adult monkeys
Mark A. Prendergast;William J. Jackson;Alvin V. Terry;Michael W. Decker.
Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors.
M.A Prendergast;B.R Harris;P.J Mullholland;J.A Blanchard.
Repeated Exposures to Subthreshold Doses of Chlorpyrifos in Rats: Hippocampal Damage, Impaired Axonal Transport, and Deficits in Spatial Learning
A. V. Terry;J. D. Stone;J. J. Buccafusco;D. W. Sickles.
Journal of Pharmacology and Experimental Therapeutics (2003)
The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors.
Barton R. Harris;D. Alex Gibson;Mark A. Prendergast;John A. Blanchard.
Alcoholism: Clinical and Experimental Research (2003)
ABT-089 (2-Methyl-3-(2-(S)-pyrrolidinylmethoxy)pyridine dihydrochloride): II. A Novel Cholinergic Channel Modulator with Effects on Cognitive Performance in Rats and Monkeys
Michael W. Decker;Anthony W. Bannon;Peter Curzon;Karen L. Gunther.
Journal of Pharmacology and Experimental Therapeutics (1997)
Fluoxetine-induced inhibition of male rat copulatory behavior: modification by lesions of the nucleus paragigantocellularis.
David P. Yells;Mark A. Prendergast;Shelton E. Hendricks;Motoyuki Nakamura.
Pharmacology, Biochemistry and Behavior (1994)
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