University of Massachusetts Medical School
His scientific interests lie mostly in Virology, Viral replication, Influenza A virus, Dengue virus and RNA interference. A large part of his Virology studies is devoted to Viral Receptor. His Viral replication research incorporates themes from Transcription, Gene and Forward genetics.
His Influenza A virus research is multidisciplinary, incorporating elements of Hemagglutinin, Interferon and Ebola virus. His Dengue virus study is focused on Virus in general. His RNA interference research includes themes of Arbovirus, Functional genomics, Flavivirus, Computational biology and Candidate gene.
Abraham L. Brass mainly focuses on Virology, Viral replication, Cell biology, Influenza A virus and Virus. Abraham L. Brass has included themes like Immunology and Microbiology in his Virology study. His Viral replication research incorporates elements of RNA, RNA interference, Reverse transcriptase, Gene and Molecular biology.
He performs integrative Influenza A virus and Membrane protein research in his work. His studies deal with areas such as Cell culture, Innate immune system and Endosome as well as Interferon. As a part of the same scientific study, Abraham L. Brass usually deals with the Innate immune system, concentrating on Antibody-dependent enhancement and frequently concerns with Immune system.
Abraham L. Brass mainly focuses on Virology, Virus, Cell biology, Viral entry and Computational biology. In his works, Abraham L. Brass undertakes multidisciplinary study on Virology and Rilpivirine. His Virus research is multidisciplinary, relying on both Interferon, Nucleus, Gene, Heterochromatin and Endosome.
His research in Cell biology intersects with topics in Gene knockout, Mechanism of action, Yeast and Sulforaphane. Abraham L. Brass interconnects Lytic cycle and Picornavirus in the investigation of issues within Viral entry. His Computational biology research incorporates themes from CRISPR, Antigenicity, Glycoprotein, Glycan and Genetic screen.
His scientific interests lie mostly in Cell biology, Capsid, Drug resistance, Dosing and Small molecule. The concepts of his Cell biology study are interwoven with issues in Virus, Lipid bilayer fusion, Human cytomegalovirus and Cell fusion. His study in Virus is interdisciplinary in nature, drawing from both Interferon and Endocytic cycle.
Abraham L. Brass has researched Capsid in several fields, including Integrase, Heterochromatin, HIV integration and Nucleus. His Drug resistance research incorporates elements of Humanized mouse, In vitro, Potency and Virology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Identification of Host Proteins Required for HIV Infection Through a Functional Genomic Screen
Abraham L. Brass;Derek M. Dykxhoorn;Yair Benita;Nan Yan.
The IFITM Proteins Mediate Cellular Resistance to Influenza A H1N1 Virus, West Nile Virus, and Dengue Virus
Abraham L. Brass;Abraham L. Brass;Abraham L. Brass;I-Chueh Huang;Yair Benita;Sinu P. John.
Meta- and Orthogonal Integration of Influenza “OMICs” Data Defines a Role for UBR4 in Virus Budding
Shashank Tripathi;Marie O. Pohl;Yingyao Zhou;Ariel Rodriguez-Frandsen.
Cell Host & Microbe (2015)
IFITM3 restricts the morbidity and mortality associated with influenza
Aaron R. Everitt;Simon Clare;Thomas Charles Pertel;Sinu P. John.
Distinct patterns of IFITM-mediated restriction of filoviruses, SARS coronavirus, and influenza A virus.
I-Chueh Huang;Charles C. Bailey;Jessica L. Weyer;Sheli R. Radoshitzky.
PLOS Pathogens (2011)
RNA interference screen for human genes associated with West Nile virus infection.
Manoj N Krishnan;Aylwin Ng;Bindu Sukumaran;Felicia D Gilfoy.
A genome-wide genetic screen for host factors required for hepatitis C virus propagation
Qisheng Li;Abraham L. Brass;Aylwin Ng;Zongyi Hu.
Proceedings of the National Academy of Sciences of the United States of America (2009)
IFITM3 Inhibits Influenza A Virus Infection by Preventing Cytosolic Entry
Eric M. Feeley;Jennifer S. Sims;Sinu P. John;Christopher R. Chin.
PLOS Pathogens (2011)
Pip, a lymphoid-restricted IRF, contains a regulatory domain that is important for autoinhibition and ternary complex formation with the Ets factor PU.1.
A L Brass;E Kehrli;C F Eisenbeis;U Storb.
Genes & Development (1996)
Identification of Zika Virus and Dengue Virus Dependency Factors using Functional Genomics
George Savidis;William M. McDougall;Paul Meraner;Jill M. Perreira.
Cell Reports (2016)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: