National Institutes of Health
United States
A. Barry Kay spends much of his time researching Immunology, Eosinophil, Interleukin 5, Mepolizumab and Asthma. His study brings together the fields of Pathology and Immunology. His work deals with themes such as Cytokine and Cell biology, which intersect with Eosinophil.
His biological study spans a wide range of topics, including Tenascin, Monoclonal, Transforming growth factor, Transforming growth factor beta and Cell type. His work on Airway hyperresponsiveness, Airway Remodelling and Airway inflammation as part of general Asthma research is often related to Mucus, thus linking different fields of science. His Immunoglobulin E research is multidisciplinary, incorporating elements of T cell and Antigen.
His primary areas of investigation include Immunology, Eosinophil, Allergy, Immunoglobulin E and Allergen. His Immunology study focuses mostly on Asthma, T cell, Cytokine, Interleukin 5 and Epitope. His Eosinophil research includes elements of Molecular biology, Basophil, Granulocyte and Pathology.
He interconnects Interleukin 10, Immunopathology and Immunotherapy in the investigation of issues within Allergy. His work carried out in the field of Immunoglobulin E brings together such families of science as Dendritic cell and Allergic inflammation. The study incorporates disciplines such as Mast cell, Eosinophilia, Inhalation and Myofibroblast in addition to Allergen.
Immunology, Allergy, Pathology, Late phase and Asthma are his primary areas of study. His Allergy study incorporates themes from Dermatology, Epitope and Immunotherapy. His research in Pathology intersects with topics in Eosinophil, Fibronectin, Matrix metalloproteinase and Allergic inflammation.
His study explores the link between Eosinophil and topics such as Staining that cross with problems in Mast cell. His Late phase research integrates issues from Omalizumab, Immunoglobulin E and Allergen challenge. His Asthma research incorporates themes from Anesthesia, Methacholine and Interleukin 5.
His main research concerns Immunology, Allergy, Allergen, Chemokine and Immunotherapy. His research on Immunology focuses in particular on Interleukin 13. His Allergy study combines topics from a wide range of disciplines, such as Dermis and Pathology.
A. Barry Kay combines subjects such as Mast cell, Specific Airway Conductance, Anesthesia and Asthma with his study of Allergen. His studies in Chemokine integrate themes in fields like Tumor necrosis factor alpha, Follistatin and Activin receptor, Transforming growth factor beta, Signal transduction. His Immunotherapy study integrates concerns from other disciplines, such as Epitope, Peptide vaccine, Cytokine secretion, Interleukin 10 and Fel d 1.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Eosinophils: Biological Properties and Role in Health and Disease
Simon P. Hogan;Helene F. Rosenberg;Redwan Moqbel;Simon Phipps.
Clinical & Experimental Allergy (2008)
Eosinophil's Role Remains Uncertain as Anti–Interleukin-5 only Partially Depletes Numbers in Asthmatic Airway
Patrick T. Flood-Page;Andrew N. Menzies-Gow;A. Barry Kay;Douglas S. Robinson.
American Journal of Respiratory and Critical Care Medicine (2003)
Anti-IL-5 treatment reduces deposition of ECM proteins in the bronchial subepithelial basement membrane of mild atopic asthmatics
Patrick Flood-Page;Andrew Menzies-Gow;Simon Phipps;Sun Ying.
Journal of Clinical Investigation (2003)
The role of T lymphocytes in the pathogenesis of asthma
Mark Larché;Douglas S. Robinson;A.Barry Kay.
The Journal of Allergy and Clinical Immunology (2003)
Enhanced expression of eotaxin and CCR3 mRNA and protein in atopic asthma. Association with airway hyperresponsiveness and predominant co-localization of eotaxin mRNA to bronchial epithelial and endothelial cells.
Sun Ying;Douglas S. Robinson;Qiu Meng;James Rottman.
European Journal of Immunology (1997)
Grass pollen immunotherapy inhibits allergen-induced infiltration of CD4+ T lymphocytes and eosinophils in the nasal mucosa and increases the number of cells expressing messenger RNA for interferon-γ
Stephen R. Durham;Sun Ying;Veronica A. Varney;Mikila R. Jacobson.
The Journal of Allergy and Clinical Immunology (1996)
The immunopathology of extrinsic (atopic) and intrinsic (non-atopic) asthma: more similarities than differences.
Marc Humbert;Günter Menz;Sun Ying;Christopher J Corrigan.
Immunology Today (1999)
A role for eosinophils in airway remodelling in asthma
A. Barry Kay;Simon Phipps;Simon Phipps;Douglas S. Robinson;Douglas S. Robinson.
Trends in Immunology (2004)
Immunoglobulin E-independent major histocompatibility complex-restricted T cell peptide epitope-induced late asthmatic reactions.
Brigitte M. Haselden;A. Barry Kay;Mark Larché.
Journal of Experimental Medicine (1999)
Anti–IL-5 (mepolizumab) therapy induces bone marrow eosinophil maturational arrest and decreases eosinophil progenitors in the bronchial mucosa of atopic asthmatics
Andrew Menzies-Gow;Patrick Flood-Page;Roma Sehmi;John Burman.
The Journal of Allergy and Clinical Immunology (2003)
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